Globotriaosylceramide leads to KCa3.1 channel dysfunction: a new insight into endothelial dysfunction in Fabry disease

Superoxide Generated by Lysophosphatidylcholine Induces Endothelial Nitric Oxide Synthase Downregulation in Human Endothelial Cells

(2010) Shinkyu Choi et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

(2009) Sebastian Brähler et al.   CIRCULATION

Extracellular K+ concentration controls cell surface density of IKr in rabbit hearts and of the HERG channel in human cell lines

(2009) Jun Guo et al.   JOURNAL OF CLINICAL INVESTIGATION

Decreased Nitric Oxide Bioavailability in a Mouse Model of Fabry Disease

(2009) L. Shu et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Apparent intermediate K conductance channel hyposmotic activation in human lens epithelial cells

(2008) Peter K. Lauf et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

VASCULAR DYSFUNCTION IN THE α-GALACTOSIDASE A-KNOCKOUT MOUSE IS AN ENDOTHELIAL CELL-, PLASMA MEMBRANE-BASED DEFECT

(2008) James L Park et al.   CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY

The Na+/Ca2+ exchanger inhibitor KB-R7943 activates large-conductance Ca2+-activated K+ channels in endothelial and vascular smooth muscle cells

(2008) Guo Hua Liang et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Fabry's disease

(2008) Yuri A Zarate et al.   LANCET

Globotriaosylceramide induces oxidative stress and up-regulates cell adhesion molecule expression in Fabry disease endothelial cells

(2008) Jin-Song Shen et al.   MOLECULAR GENETICS AND METABOLISM