Advanced glycation endproducts trigger autophagy in cadiomyocyte Via RAGE/PI3K/AKT/mTOR pathway
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Title
Advanced glycation endproducts trigger autophagy in cadiomyocyte Via RAGE/PI3K/AKT/mTOR pathway
Authors
Keywords
Cardiomyocyte, Autophagy, Advanced glycation end products, Hear function, Signal pathway
Journal
Cardiovascular Diabetology
Volume 13, Issue 1, Pages 78
Publisher
Springer Nature
Online
2014-04-14
DOI
10.1186/1475-2840-13-78
References
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Related references
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- Caffeine induces apoptosis by enhancement of autophagy via PI3K/Akt/mTOR/p70S6K inhibition
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- Pre-clinical diabetic cardiomyopathy: prevalence, screening, and outcome
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- Role of autophagy in heart failure associated with aging
- (2010) Guido R. Y. De Meyer et al. HEART FAILURE REVIEWS
- Advanced glycation end products impair function of late endothelial progenitor cells through effects on protein kinase Akt and cyclooxygenase-2
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- Understanding Autophagy in Cell Death Control
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- Crosstalk Between Autophagy and Apoptosis in Heart Disease
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- Autophagy in Load-Induced Heart Disease
- (2008) Beverly A. Rothermel et al. CIRCULATION RESEARCH
- The Role of AGEs in Cardiovascular Disease
- (2008) Karin Jandeleit-Dahm et al. CURRENT PHARMACEUTICAL DESIGN
- Cell death and autophagy: Cytokines, drugs, and nutritional factors
- (2008) Wilfried Bursch et al. TOXICOLOGY
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