ROS-independent JNK activation and multisite phosphorylation of Bcl-2 link diallyl tetrasulfide-induced mitotic arrest to apoptosis
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
ROS-independent JNK activation and multisite phosphorylation of Bcl-2 link diallyl tetrasulfide-induced mitotic arrest to apoptosis
Authors
Keywords
-
Journal
CARCINOGENESIS
Volume 33, Issue 11, Pages 2162-2171
Publisher
Oxford University Press (OUP)
Online
2012-07-22
DOI
10.1093/carcin/bgs240
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Chemical Properties and Mechanisms Determining the Anti-Cancer Action of Garlic-Derived Organic Sulfur Compounds
- (2012) Claudia Cerella et al. Anti-Cancer Agents in Medicinal Chemistry
- COX-2 inhibitors block chemotherapeutic agent-induced apoptosis prior to commitment in hematopoietic cancer cells
- (2011) Claudia Cerella et al. BIOCHEMICAL PHARMACOLOGY
- Integrin-Dependent Activation of the JNK Signaling Pathway by Mechanical Stress
- (2011) Andrea Maria Pereira et al. PLoS One
- Interactions of polysulfanes with components of red blood cells
- (2011) Thomas Schneider et al. MedChemComm
- UNBS1450, a steroid cardiac glycoside inducing apoptotic cell death in human leukemia cells
- (2010) Tom Juncker et al. BIOCHEMICAL PHARMACOLOGY
- The c-Jun N-terminal kinase JNK functions upstream of Aurora B to promote entry into mitosis
- (2010) Kutluk Oktay et al. CELL CYCLE
- Allyl Sulfides Inhibit Cell Growth of Skin Cancer Cells through Induction of DNA Damage Mediated G2/M Arrest and Apoptosis
- (2010) Hsiao−Chi Wang et al. JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY
- Role of Bim in diallyl trisulfide-induced cytotoxicity in human cancer cells
- (2010) Byeong-Chel Lee et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Multistep and multitask Bax activation
- (2010) Lina Ghibelli et al. MITOCHONDRION
- Cell cycle arrest in early mitosis and induction of caspase-dependent apoptosis in U937 cells by diallyltetrasulfide (Al2S4)
- (2009) Claudia Cerella et al. APOPTOSIS
- Heteronemin, a spongean sesterterpene, inhibits TNFα-induced NF-κB activation through proteasome inhibition and induces apoptotic cell death
- (2009) Marc Schumacher et al. BIOCHEMICAL PHARMACOLOGY
- Apoptosis induction in human lung adenocarcinoma cells by oil-soluble allyl sulfides: Triggers, pathways, and modulators
- (2009) Xin-Jiang Wu et al. ENVIRONMENTAL AND MOLECULAR MUTAGENESIS
- Molecular mechanism of diallyl disulfide in cell cycle arrest and apoptosis in HCT-116 colon cancer cells
- (2009) Ju-Dong Song et al. JOURNAL OF BIOCHEMICAL AND MOLECULAR TOXICOLOGY
- Potential role of organic sulfur compounds from Allium species in cancer prevention and therapy
- (2009) C. Scherer et al. PHYTOCHEMISTRY REVIEWS
- Apoptosis induced by diallyl disulfide in human breast cancer cell line MCF-71
- (2008) Xiao-yong LEI et al. ACTA PHARMACOLOGICA SINICA
- Reactive oxygen species (ROS) in multistage carcinogenesis
- (2008) Mihalis Panayiotidis CANCER LETTERS
- Multitargeted prevention and therapy of cancer by diallyl trisulfide and related Allium vegetable-derived organosulfur compounds
- (2008) Anna A. Powolny et al. CANCER LETTERS
- Alkenyl group is responsible for the disruption of microtubule network formation in human colon cancer cell line HT-29 cells
- (2008) T. Hosono et al. CARCINOGENESIS
- Diallyl disulfide induces apoptosis in human colon cancer cell line (COLO 205) through the induction of reactive oxygen species, endoplasmic reticulum stress, caspases casade and mitochondrial-dependent pathways
- (2008) Jai-Sing Yang et al. FOOD AND CHEMICAL TOXICOLOGY
- Microtubule assembly dynamics: An attractive target for anticancer drugs
- (2008) Parminder Singh et al. IUBMB LIFE
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started