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Title
Targets of Raf in tumorigenesis
Authors
Keywords
-
Journal
CARCINOGENESIS
Volume 31, Issue 7, Pages 1165-1174
Publisher
Oxford University Press (OUP)
Online
2010-01-05
DOI
10.1093/carcin/bgp337
References
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Related references
Note: Only part of the references are listed.- Constitutive Activation of Raf-1 Induces Glioma Formation in Mice
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- The Role of BRAF Mutation and p53 Inactivation during Transformation of a Subpopulation of Primary Human Melanocytes
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- Sprouty2 Association with B-Raf Is Regulated by Phosphorylation and Kinase Conformation
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- Sorafenib Inhibits Non-Small Cell Lung Cancer Cell Growth by Targeting B-RAF in KRAS Wild-Type Cells and C-RAF in KRAS Mutant Cells
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- Antitumor Efficacy of the Novel RAF Inhibitor GDC-0879 Is Predicted by BRAFV600E Mutational Status and Sustained Extracellular Signal-Regulated Kinase/Mitogen-Activated Protein Kinase Pathway Suppression
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- C-Raf Is Associated with Disease Progression and Cell Proliferation in a Subset of Melanomas
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- The H3K27me3 demethylase JMJD3 contributes to the activation of the INK4A-ARF locus in response to oncogene- and stress-induced senescence
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- Identification of Novelin VivoPhosphorylation Sites of the Human Proapoptotic Protein BAD
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- Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1
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- Requirement for NF-κB signalling in a mouse model of lung adenocarcinoma
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- BrafV600E cooperates with Pten loss to induce metastatic melanoma
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- A Mek1–Mek2 heterodimer determines the strength and duration of the Erk signal
- (2009) Federica Catalanotti et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
- Plexin B1 is repressed by oncogenic B-Raf signaling and functions as a tumor suppressor in melanoma cells
- (2009) G M Argast et al. ONCOGENE
- Oncogenic RAF1 rearrangement and a novel BRAF mutation as alternatives to KIAA1549:BRAF fusion in activating the MAPK pathway in pilocytic astrocytoma
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- BRAF gene duplication constitutes a mechanism of MAPK pathway activation in low-grade astrocytomas
- (2008) Stefan Pfister et al. JOURNAL OF CLINICAL INVESTIGATION
- AberrantBRAFsplicing as an alternative mechanism for oncogenic B-Raf activation in thyroid carcinoma
- (2008) Essa Y Baitei et al. JOURNAL OF PATHOLOGY
- Essential Role of ERK Dimers in the Activation of Cytoplasmic but Not Nuclear Substrates by ERK-Scaffold Complexes
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- CRAF Autophosphorylation of Serine 621 Is Required to Prevent Its Proteasome-Mediated Degradation
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- CRAF inhibition induces apoptosis in melanoma cells with non-V600E BRAF mutations
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- B-Raf is required for ERK activation and tumor progression in a mouse model of pancreatic β-cell carcinogenesis
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- C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells
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- Discovery of a selective inhibitor of oncogenic B-Raf kinase with potent antimelanoma activity
- (2008) J. Tsai et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- B-RAF Regulation of Rnd3 Participates in Actin Cytoskeletal and Focal Adhesion Organization
- (2007) R. Matthew Klein et al. MOLECULAR BIOLOGY OF THE CELL
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