Inhibition of N-linked glycosylation by tunicamycin induces E-cadherin-mediated cell–cell adhesion and inhibits cell proliferation in undifferentiated human colon cancer cells

Hypoglycosylated E-cadherin promotes the assembly of tight junctions through the recruitment of PP2A to adherens junctions

(2010) Mihai Nita-Lazar et al.   EXPERIMENTAL CELL RESEARCH

Role of E-cadherin N-glycosylation profile in a mammary tumor model

(2009) Salomé S. Pinho et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Prostaglandin E2-EP1 and EP2 receptor signaling promotes apical junctional complex disassembly of Caco-2 human colorectal cancer cells

(2009) Marcelo N Tanaka et al.   BMC CELL BIOLOGY

Activation of ER stress and inhibition of EGFR N-glycosylation by tunicamycin enhances susceptibility of human non-small cell lung cancer cells to erlotinib

(2009) Yi-He Ling et al.   CANCER CHEMOTHERAPY AND PHARMACOLOGY

Overexpression of DPAGT1 Leads to Aberrant N-Glycosylation of E-Cadherin and Cellular Discohesion in Oral Cancer

(2009) M. Nita-Lazar et al.   CANCER RESEARCH

The role of N-acetylglucosaminyltransferase III and V in the post-transcriptional modifications of E-cadherin

(2009) S. S. Pinho et al.   HUMAN MOLECULAR GENETICS

Inhibition of N-Linked Glycosylation Disrupts Receptor Tyrosine Kinase Signaling in Tumor Cells

(2008) J. N. Contessa et al.   CANCER RESEARCH

Organization of multiprotein complexes at cell–cell junctions

(2008) Klaus Ebnet   HISTOCHEMISTRY AND CELL BIOLOGY

A Cross-Link between Protein Kinase A and Rho-Family GTPases Signaling Mediates Cell-Cell Adhesion and Actin Cytoskeleton Organization in Epithelial Cancer Cells

(2008) F. Leve et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS