HIF-1α-independent hypoxia-induced rapid PTK6 stabilization is associated with increased motility and invasion
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Title
HIF-1α-independent hypoxia-induced rapid PTK6 stabilization is associated with increased motility and invasion
Authors
Keywords
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Journal
CANCER BIOLOGY & THERAPY
Volume 15, Issue 10, Pages 1350-1357
Publisher
Informa UK Limited
Online
2014-07-15
DOI
10.4161/cbt.29822
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- (2013) Shin-Ae Kang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
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- (2012) Shin-Ae Kang et al. BIOCHEMICAL JOURNAL
- Targeting radiation-resistant hypoxic tumour cells through ATR inhibition
- (2012) I M Pires et al. BRITISH JOURNAL OF CANCER
- Hypoxia-Inducible Factors in Physiology and Medicine
- (2012) Gregg L. Semenza CELL
- Suppressor of Cytokine Signaling 3 Inhibits Breast Tumor Kinase Activation of STAT3
- (2012) Yiwei Gao et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Protein tyrosine kinase 6 protects cells from anoikis by directly phosphorylating focal adhesion kinase and activating AKT
- (2012) Y Zheng et al. ONCOGENE
- High-resolution genome-wide mapping of HIF-binding sites by ChIP-seq
- (2011) J. Schodel et al. BLOOD
- Hypoxia-driven cell motility reflects the interplay between JMY and HIF-1α
- (2011) A S Coutts et al. ONCOGENE
- The von Hippel-Lindau Tumor Suppressor Protein Promotes c-Cbl-Independent Poly-Ubiquitylation and Degradation of the Activated EGFR
- (2011) Liang Zhou et al. PLoS One
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- (2010) M Aubele et al. BRITISH JOURNAL OF CANCER
- Effects of Acute versus Chronic Hypoxia on DNA Damage Responses and Genomic Instability
- (2010) I. M. Pires et al. CANCER RESEARCH
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- (2010) Patrick M. Brauer et al. CELL CYCLE
- Hypoxia and Hypoxia-Inducible Factors: Master Regulators of Metastasis
- (2010) X. Lu et al. CLINICAL CANCER RESEARCH
- Brk/PTK6 signaling in normal and cancer cell models
- (2010) Julie H Ostrander et al. CURRENT OPINION IN PHARMACOLOGY
- PTK6 Regulates IGF-1-Induced Anchorage-Independent Survival
- (2010) Hanna Y. Irie et al. PLoS One
- Brk Protects Breast Cancer Cells from Autophagic Cell Death Induced by Loss of Anchorage
- (2009) Amanda J. Harvey et al. AMERICAN JOURNAL OF PATHOLOGY
- An online survival analysis tool to rapidly assess the effect of 22,277 genes on breast cancer prognosis using microarray data of 1,809 patients
- (2009) Balazs Györffy et al. BREAST CANCER RESEARCH AND TREATMENT
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- (2009) Andrea Haegebarth et al. GASTROENTEROLOGY
- Genome-wide Association of Hypoxia-inducible Factor (HIF)-1α and HIF-2α DNA Binding with Expression Profiling of Hypoxia-inducible Transcripts
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- (2009) H. L. Palka-Hamblin et al. JOURNAL OF CELL SCIENCE
- Osteopontin Promotes Vascular Endothelial Growth Factor Dependent Breast Tumor Growth and Angiogenesis via Autocrine and Paracrine Mechanisms
- (2008) G. Chakraborty et al. CANCER RESEARCH
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