Effects of the antianginal drug, ranolazine, on the brain sodium channel NaV1.2 and its modulation by extracellular protons

Acidosis Differentially Modulates Inactivation in NaV1.2, NaV1.4, and NaV1.5 Channels

(2012) Yury Y. Vilin et al.   Frontiers in Pharmacology

Guide to Receptors and Channels (GRAC), 5th edition

(2011) SPH Alexander et al.   BRITISH JOURNAL OF PHARMACOLOGY

SCN1A splice variants exhibit divergent sensitivity to commonly used antiepileptic drugs

(2011) Christopher H. Thompson et al.   EPILEPSIA

Ranolazine selectively blocks persistent current evoked by epilepsy-associated NaV1.1 mutations

(2010) Kristopher M Kahlig et al.   BRITISH JOURNAL OF PHARMACOLOGY

Block of Tetrodotoxin-sensitive, Nav1.7, and Tetrodotoxin-resistant, Nav1.8, Na+channels by Ranolazine

(2010) Sridharan Rajamani et al.   Channels

The Anti-Ischemic Mechanism of Action of Ranolazine in Stable Ischemic Heart Disease

(2010) Peter H. Stone et al.   JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY

Voltage-gated sodium channels as therapeutic targets in epilepsy and other neurological disorders

(2010) Massimo Mantegazza et al.   LANCET NEUROLOGY

Effects of Ranolazine on Wild-Type and Mutant hNav1.7 Channels and on DRG Neuron Excitability

(2010) Mark Estacion et al.   Molecular Pain

Rapid Kinetic Interactions of Ranolazine With HERG K+ Current

(2009) Sridharan Rajamani et al.   JOURNAL OF CARDIOVASCULAR PHARMACOLOGY

Ranolazine Shortens Repolarization in Patients with Sustained Inward Sodium Current Due to Type-3 Long-QT Syndrome

(2008) ARTHUR J. MOSS et al.   JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY

Ranolazine improves diastolic dysfunction in isolated myocardium from failing human hearts — Role of late sodium current and intracellular ion accumulation

(2008) Samuel Sossalla et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY