CD22 Exon 12 deletion is a characteristic genetic defect of therapy-refractory clones in paediatric acute lymphoblastic leukaemia

JAK3 pathway is constitutively active in B-lineage acute lymphoblastic leukemia

(2010) Fatih M Uckun et al.   Expert Review of Anticancer Therapy

Genome-wide expression analysis of paired diagnosis–relapse samples in ALL indicates involvement of pathways related to DNA replication, cell cycle and DNA repair, independent of immune phenotype

(2010) F J T Staal et al.   LEUKEMIA

STAT3 is a substrate of SYK tyrosine kinase in B-lineage leukemia/lymphoma cells exposed to oxidative stress

(2010) F. M. Uckun et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

CD22 EXON 12 deletion as a pathogenic mechanism of human B-precursor leukemia

(2010) F. M. Uckun et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Novel roles of U1 snRNP in alternative splicing regulation

(2010) Emanuele Buratti et al.   RNA Biology

Fluorescent Probes for Live-Cell RNA Detection

(2009) Gang Bao et al.   Annual Review of Biomedical Engineering

Gene silencing by synthetic U1 Adaptors

(2009) Rafal Goraczniak et al.   NATURE BIOTECHNOLOGY

Bone-marrow relapse in paediatric acute lymphoblastic leukaemia

(2008) L Charles Bailey et al.   LANCET ONCOLOGY