ERRβ signalling through FST and BCAS2 inhibits cellular proliferation in breast cancer cells

Dishonorable Discharge: The Oncogenic Roles of Cleaved E-Cadherin Fragments

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Molecular Classification of Estrogen Receptor-positive/Luminal Breast Cancers

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Accelerating Protein Docking in ZDOCK Using an Advanced 3D Convolution Library

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Orphan nuclear receptors in breast cancer pathogenesis and therapeutic response

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Synergistic epigenetic reactivation of estrogen receptor-alpha (ERalpha) by combined green tea polyphenol and histone deacetylase inhibitor in ERalpha-negative breast cancer cells

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I-TASSER: a unified platform for automated protein structure and function prediction

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E-cadherin and β-catenin Loss of Expression Related to Bone Metastasis in Prostate Cancer

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Cleavage of E-Cadherin by Matrix Metalloproteinase-7 Promotes Cellular Proliferation in Nontransformed Cell Lines via Activation of RhoA

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An online survival analysis tool to rapidly assess the effect of 22,277 genes on breast cancer prognosis using microarray data of 1,809 patients

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Breast Cancer Amplified Sequence 2, a Novel Negative Regulator of the p53 Tumor Suppressor

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Selective androgen receptor modulators as function promoting therapies

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Laminin-1 induces E-cadherin expression in 3-dimensional cultured breast cancer cells by inhibiting DNA methyltransferase 1 and reversing promoter methylation status

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Modulation of ERα transcriptional activity by the orphan nuclear receptor ERRβ and evidence for differential effects of long- and short-form splice variants☆

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A SNAIL1–SMAD3/4 transcriptional repressor complex promotes TGF-β mediated epithelial–mesenchymal transition

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MCF-7 breast carcinoma cells do not express caspase-3

(2008) Reiner U. Jänicke   BREAST CANCER RESEARCH AND TREATMENT

Loss of E-Cadherin Promotes Metastasis via Multiple Downstream Transcriptional Pathways

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ERRγ Mediates Tamoxifen Resistance in Novel Models of Invasive Lobular Breast Cancer

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Follistatin as an Inhibitor of Experimental Metastasis

(2008) J. E. Talmadge   CLINICAL CANCER RESEARCH

Regulation of Myogenic Differentiation by Androgens: Cross Talk between Androgen Receptor/ β-Catenin and Follistatin/Transforming Growth Factor-β Signaling Pathways

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