MiR-204 inhibits human NSCLC metastasis through suppression of NUAK1
Published 2014 View Full Article
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Title
MiR-204 inhibits human NSCLC metastasis through suppression of NUAK1
Authors
Keywords
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Journal
BRITISH JOURNAL OF CANCER
Volume 111, Issue 12, Pages 2316-2327
Publisher
Springer Nature
Online
2014-11-21
DOI
10.1038/bjc.2014.580
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- (2013) Zhiyu Chen et al. Molecular Cancer
- DIXDC1 increases the invasion and migration ability of non-small-cell lung cancer cells via the PI3K-AKT/AP-1 pathway
- (2013) Zhonghai Xu et al. MOLECULAR CARCINOGENESIS
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- (2012) Z. Ying et al. CANCER RESEARCH
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- (2012) J. Cai et al. CANCER RESEARCH
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- (2012) Géraldine Pignot et al. INTERNATIONAL JOURNAL OF CANCER
- MicroRNA-204 critically regulates carcinogenesis in malignant peripheral nerve sheath tumors
- (2012) Meng Gong et al. NEURO-ONCOLOGY
- Genomic Loss of Tumor Suppressor miRNA-204 Promotes Cancer Cell Migration and Invasion by Activating AKT/mTOR/Rac1 Signaling and Actin Reorganization
- (2012) J. Saadi Imam et al. PLoS One
- miR-204 targets Bcl-2 expression and enhances responsiveness of gastric cancer
- (2012) A Sacconi et al. Cell Death & Disease
- Global cancer statistics
- (2011) Ahmedin Jemal et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Dual inhibition of phosphatidylinositol 3-kinase/Akt and mammalian target of rapamycin signaling in human nonsmall cell lung cancer cells by a dietary flavonoid fisetin
- (2011) Naghma Khan et al. INTERNATIONAL JOURNAL OF CANCER
- Dysregulation of microRNA-204 mediates migration and invasion of endometrial cancer by regulating FOXC1
- (2011) T.K.H. Chung et al. INTERNATIONAL JOURNAL OF CANCER
- ARK5 is associated with the invasive and metastatic potential of human breast cancer cells
- (2011) Xin-Zhong Chang et al. JOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY
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- (2011) David P. Turner et al. PROSTATE
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- (2010) F. Zhang et al. CANCER RESEARCH
- Epigenetic Silencing of miR-137 Is an Early Event in Colorectal Carcinogenesis
- (2010) F. Balaguer et al. CANCER RESEARCH
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- (2010) Priscilla F. McAuliffe et al. Clinical Breast Cancer
- An Emerging Role for Epigenetic Dysregulation in Arsenic Toxicity and Carcinogenesis
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- Rictor regulates MMP-9 activity and invasion through Raf-1-MEK-ERK signaling pathway in glioma cells
- (2010) Gowry Das et al. MOLECULAR CARCINOGENESIS
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- (2010) Younghee Lee et al. PLoS Computational Biology
- MicroRNAs in Cancer
- (2009) Ramiro Garzon et al. Annual Review of Medicine
- On the Role of Cell Signaling Models in Cancer Research
- (2009) A. C. Ventura et al. CANCER RESEARCH
- MicroRNAs and Cancer: Short RNAs Go a Long Way
- (2009) Andrea Ventura et al. CELL
- Akt2 is required for macrophage chemotaxis
- (2009) Baogang Zhang et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Reduction of Akt2 inhibits migration and invasion of glioma cells
- (2009) Baogang Zhang et al. INTERNATIONAL JOURNAL OF CANCER
- A high throughput experimental approach to identify miRNA targets in human cells
- (2009) Lu Ping Tan et al. NUCLEIC ACIDS RESEARCH
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- (2008) G. Z. Cheng et al. CANCER RESEARCH
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- (2008) K.-i. Kozaki et al. CANCER RESEARCH
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