High miR-26a and low CDC2 levels associate with decreased EZH2 expression and with favorable outcome on tamoxifen in metastatic breast cancer

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MicroRNAs: Synthesis, mechanism, function, and recent clinical trials

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Decreased expression of EZH2 is associated with upregulation of ER and favorable outcome to tamoxifen in advanced breast cancer

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MicroRNA-30c expression level is an independent predictor of clinical benefit of endocrine therapy in advanced estrogen receptor positive breast cancer

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Cyclin E Deregulation Impairs Mitotic Progression through Premature Activation of Cdc25C

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MicroRNA-101 negatively regulates Ezh2 and its expression is modulated by androgen receptor and HIF-1α/HIF-1β

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CDK1-dependent phosphorylation of EZH2 suppresses methylation of H3K27 and promotes osteogenic differentiation of human mesenchymal stem cells

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Cyclin-dependent kinases regulate epigenetic gene silencing through phosphorylation of EZH2

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Therapeutic microRNA Delivery Suppresses Tumorigenesis in a Murine Liver Cancer Model

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Subtypes of Breast Cancer Show Preferential Site of Relapse

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MicroRNA-26a Targets the Histone MethyltransferaseEnhancer of Zeste homolog 2during Myogenesis

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MicroRNA-221/222 Confers Tamoxifen Resistance in Breast Cancer by Targeting p27Kip1

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Small is beautiful: microRNAs and breast cancer-where are we now?

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Ezh1 and Ezh2 Maintain Repressive Chromatin through Different Mechanisms

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Downregulation of EZH2 decreases growth of estrogen receptor-negative invasive breast carcinoma and requires BRCA1

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Four miRNAs associated with aggressiveness of lymph node-negative, estrogen receptor-positive human breast cancer

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Genomic Loss of microRNA-101 Leads to Overexpression of Histone Methyltransferase EZH2 in Cancer

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