Thrombin inhibits HMGB1-mediated proinflammatory signaling responses when endothelial protein C receptor is occupied by its natural ligand

Role of high mobility group box 1 in inflammatory disease: Focus on sepsis

(2012) Jong-Sup Bae   ARCHIVES OF PHARMACAL RESEARCH

Activated protein C inhibits high mobility group box 1 signaling in endothelial cells

(2011) J.-S. Bae et al.   BLOOD

HMGB1 induces human lung endothelial cell cytoskeletal rearrangement and barrier disruption

(2010) Rachel K. Wolfson et al.   MICROVASCULAR RESEARCH

HMGB1 Enhances the Proinflammatory Activity of Lipopolysaccharide by Promoting the Phosphorylation of MAPK p38 through Receptor for Advanced Glycation End Products

(2009) Y.-H. Qin et al.   JOURNAL OF IMMUNOLOGY

NOVEL HMGB1-INHIBITING THERAPEUTIC AGENTS FOR EXPERIMENTAL SEPSIS

(2009) Haichao Wang et al.   SHOCK

Thrombin and activated protein C inhibit the expression of secretory group IIA phospholipase A2 in the TNF-α-activated endothelial cells by EPCR and PAR-1 dependent mechanisms

(2009) Jong-Sup Bae et al.   THROMBOSIS RESEARCH

Advanced glycation end products depress function of endothelial progenitor cells via p38 and ERK 1/2 mitogen-activated protein kinase pathways

(2008) Chengbo Sun et al.   BASIC RESEARCH IN CARDIOLOGY