Hemostatic disorders in a JAK2V617F-driven mouse model of myeloproliferative neoplasm
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Title
Hemostatic disorders in a JAK2V617F-driven mouse model of myeloproliferative neoplasm
Authors
Keywords
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Journal
BLOOD
Volume 124, Issue 7, Pages 1136-1145
Publisher
American Society of Hematology
Online
2014-06-21
DOI
10.1182/blood-2013-10-530832
References
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Note: Only part of the references are listed.- JAK2V617F-positive endothelial cells contribute to clotting abnormalities in myeloproliferative neoplasms
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- Myeloproliferative neoplasm induced by constitutive expression of JAK2V617F in knock-in mice
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- Conditional expression of heterozygous or homozygous Jak2V617F from its endogenous promoter induces a polycythemia vera-like disease
- (2010) H. Akada et al. BLOOD
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- Platelet turnover, coagulation factors, and soluble markers of platelet and endothelial activation in essential thrombocythemia: Relationship with thrombosis occurrence andJAK2 V617F allele burden
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- Recurrent thrombosis in patients with polycythemia vera and essential thrombocythemia: incidence, risk factors, and effect of treatments
- (2008) V. De Stefano et al. HAEMATOLOGICA
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