Plasmodium falciparum uses a key functional site in complement receptor type-1 for invasion of human erythrocytes

Plasmodium falciparum field isolates use complement receptor 1 (CR1) as a receptor for invasion of erythrocytes

(2011) Gordon A. Awandare et al.   MOLECULAR AND BIOCHEMICAL PARASITOLOGY

Ligation of complement receptor 1 increases erythrocyte membrane deformability

(2010) A. M. Glodek et al.   BLOOD

Erythrocyte Invasion and Merozoite Ligand Gene Expression in Severe and MildPlasmodium falciparumMalaria

(2010) Natalia Gomez‐Escobar et al.   JOURNAL OF INFECTIOUS DISEASES

Complement receptor 1 is the host erythrocyte receptor for Plasmodium falciparum PfRh4 invasion ligand

(2010) W.-H. Tham et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Production of biologically active complement factor H in therapeutically useful quantities

(2010) Christoph Q. Schmidt et al.   PROTEIN EXPRESSION AND PURIFICATION

Complement Receptor 1 Is a Sialic Acid-Independent Erythrocyte Receptor of Plasmodium falciparum

(2010) Carmenza Spadafora et al.   PLoS Pathogens

Polymorphisms in Erythrocyte Binding Antigens 140 and 181 Affect Function and Binding but Not Receptor Specificity in Plasmodium falciparum

(2009) A. G. Maier et al.   INFECTION AND IMMUNITY

Antibodies to Reticulocyte Binding Protein-Like Homologue 4 Inhibit Invasion of Plasmodium falciparum into Human Erythrocytes

(2009) W.-H. Tham et al.   INFECTION AND IMMUNITY

Structure of complement fragment C3b–factor H and implications for host protection by complement regulators

(2009) Jin Wu et al.   NATURE IMMUNOLOGY

Glycophorin B is the erythrocyte receptor of Plasmodium falciparum erythrocyte-binding ligand, EBL-1

(2009) D. C. G. Mayer et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Morphology and kinetics of the three distinct phases of red blood cell invasion by Plasmodium falciparum merozoites

(2008) Paul R. Gilson et al.   INTERNATIONAL JOURNAL FOR PARASITOLOGY

Variation in use of erythrocyte invasion pathways by Plasmodium falciparum mediates evasion of human inhibitory antibodies

(2008) Kristina E.M. Persson et al.   JOURNAL OF CLINICAL INVESTIGATION