Hyperproliferation, cancer, and inflammation in mice expressing a 133p53-like isoform
Published 2011 View Full Article
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Title
Hyperproliferation, cancer, and inflammation in mice expressing a 133p53-like isoform
Authors
Keywords
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Journal
BLOOD
Volume 117, Issue 19, Pages 5166-5177
Publisher
American Society of Hematology
Online
2011-03-17
DOI
10.1182/blood-2010-11-321851
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Note: Only part of the references are listed.- p53 directly transactivates Δ133p53α, regulating cell fate outcome in response to DNA damage
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- (2009) J. Chen et al. GENES & DEVELOPMENT
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- Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources
- (2009) Da Wei Huang et al. Nature Protocols
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- (2008) K. A. Avery-Kiejda et al. CLINICAL CANCER RESEARCH
- An interferon-related gene signature for DNA damage resistance is a predictive marker for chemotherapy and radiation for breast cancer
- (2008) R. R. Weichselbaum et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Knockin mice expressing a chimeric p53 protein reveal mechanistic differences in how p53 triggers apoptosis and senescence
- (2008) T. M. Johnson et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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