Sirt1 Regulates Microtubule Dynamics Through Negative Regulation of Plk1 in Mitosis
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Title
Sirt1 Regulates Microtubule Dynamics Through Negative Regulation of Plk1 in Mitosis
Authors
Keywords
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Journal
JOURNAL OF CELLULAR BIOCHEMISTRY
Volume 116, Issue 9, Pages 1888-1897
Publisher
Wiley
Online
2015-03-04
DOI
10.1002/jcb.25144
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- HIV-1 Tat impairs cell cycle control by targeting the Tip60, Plk1 and cyclin B1 ternary complex
- (2012) Shi-Meng Zhang et al. CELL CYCLE
- The Spindle Assembly Checkpoint
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- SIRT1, Is It a Tumor Promoter or Tumor Suppressor?
- (2012) Chu-Xia Deng International Journal of Biological Sciences
- Human SIRT1 associates with mitotic chromatin and contributes to chromosomal condensation
- (2011) Samuel T. Fatoba et al. CELL CYCLE
- Generation of Cancerous Neural Stem Cells Forming Glial Tumor by Oncogenic Stimulation
- (2011) Ji-Seon Lee et al. Stem Cell Reviews and Reports
- SIRT Inhibitors Induce Cell Death and p53 Acetylation through Targeting Both SIRT1 and SIRT2
- (2010) Barrie Peck et al. MOLECULAR CANCER THERAPEUTICS
- ICIS and Aurora B Coregulate the Microtubule Depolymerase Kif2a
- (2009) Anne L. Knowlton et al. CURRENT BIOLOGY
- Plk1 and Aurora A regulate the depolymerase activity and the cellular localization of Kif2a
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- Impaired DNA Damage Response, Genome Instability, and Tumorigenesis in SIRT1 Mutant Mice
- (2008) Rui-Hong Wang et al. CANCER CELL
- DDA3 recruits microtubule depolymerase Kif2a to spindle poles and controls spindle dynamics and mitotic chromosome movement
- (2008) Chang-Young Jang et al. JOURNAL OF CELL BIOLOGY
- Polo-like kinase-1 is activated by aurora A to promote checkpoint recovery
- (2008) Libor Macůrek et al. NATURE
- Phosphorylation of Plk1 at Ser326 regulates its functions during mitotic progression
- (2008) J Tang et al. ONCOGENE
- Opposing Effects of Sirtuins on Neuronal Survival: SIRT1-Mediated Neuroprotection Is Independent of Its Deacetylase Activity
- (2008) Jason A. Pfister et al. PLoS One
- Phosphorylation Regulates SIRT1 Function
- (2008) Tsutomu Sasaki et al. PLoS One
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