Human Sperm Devoid of Germinal Angiotensin-Converting Enzyme Is Responsible for Total Fertilization Failure and Lower Fertilization Rates by Conventional In Vitro Fertilization1

A Novel Splice-Site Mutation in Angiotensin I-Converting Enzyme (ACE) Gene, c.3691+1G>A (IVS25+1G>A), Causes a Dramatic Increase in Circulating ACE through Deletion of the Transmembrane Anchor

(2013) Alexandre Persu et al.   PLoS One

Expression of TEX101, regulated by ACE, is essential for the production of fertile mouse spermatozoa

(2013) Y. Fujihara et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Mechanisms of Fertilization--A View From the Study of Gene-Manipulated Mice

(2011) Y. Muro et al.   JOURNAL OF ANDROLOGY

Total fertilization failure and idiopathic subfertility

(2009) Sandra J Tanahatoe et al.   Reproductive Biology and Endocrinology

A Common Protamine 1 Promoter Polymorphism (-190 C->A) Correlates With Abnormal Sperm Morphology and Increased Protamine P1/P2 Ratio in Infertile Patients

(2008) C. Gazquez et al.   JOURNAL OF ANDROLOGY

Human sperm devoid of PLC, zeta 1 fail to induce Ca2+ release and are unable to initiate the first step of embryo development

(2008) Sook-Young Yoon et al.   JOURNAL OF CLINICAL INVESTIGATION

Epitope mapping of mAbs to denatured human testicular ACE (CD143)

(2008) I. V. Balyasnikova et al.   TISSUE ANTIGENS