The Disturbances of Endoplasmic Reticulum Calcium Homeostasis Caused by Increased Intracellular Reactive Oxygen Species Contributes to Fragmentation in Aged Porcine Oocytes1

Maternal diabetes causes abnormal dynamic changes of endoplasmic reticulum during mouse oocyte maturation and early embryo development

(2013) Chun-Hui Zhang et al.   Reproductive Biology and Endocrinology

Follicle-stimulating Hormone Regulates Pro-apoptotic Protein Bcl-2-interacting Mediator of Cell Death-Extra Long (Bim EL )-induced Porcine Granulosa Cell Apoptosis

(2012) Xian-Long Wang et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

PML Regulates Apoptosis at Endoplasmic Reticulum by Modulating Calcium Release

(2010) C. Giorgi et al.   SCIENCE

Acetylation of H4K12 in porcine oocytes during in vitro aging: potential role of ooplasmic reactive oxygen species

(2010) Mao-Sheng Cui et al.   THERIOGENOLOGY

Abnormal changes in mitochondria, lipid droplets, ATP and glutathione content, and Ca2+release after electro-activation contribute to poor developmental competence of porcine oocyte duringin vitroageing

(2009) Ze-Dong Hao et al.   REPRODUCTION FERTILITY AND DEVELOPMENT

Calcium and apoptosis: ER-mitochondria Ca2+ transfer in the control of apoptosis

(2008) P Pinton et al.   ONCOGENE

Reactive oxygen species and oocyte aging: Role of superoxide, hydrogen peroxide, and hypochlorous acid

(2007) Anuradha P. Goud et al.   FREE RADICAL BIOLOGY AND MEDICINE