Curcumin improves hypoxia induced dysfunctions in 3T3-L1 adipocytes by protecting mitochondria and down regulating inflammation
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Curcumin improves hypoxia induced dysfunctions in 3T3-L1 adipocytes by protecting mitochondria and down regulating inflammation
Authors
Keywords
-
Journal
BIOFACTORS
Volume 40, Issue 5, Pages 513-523
Publisher
Wiley
Online
2014-08-11
DOI
10.1002/biof.1175
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- ATF3 plays a role in adipocyte hypoxia-mediated mitochondria dysfunction in obesity
- (2013) Min-Kyung Jang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Resveratrol has inhibitory effects on the hypoxia-induced inflammation and angiogenesis in human adipose tissue in vitro
- (2013) Karina B. Cullberg et al. EUROPEAN JOURNAL OF PHARMACEUTICAL SCIENCES
- Hypoxia and Adipose Tissue Function and Dysfunction in Obesity
- (2013) Paul Trayhurn PHYSIOLOGICAL REVIEWS
- Hypoxia-inducible Factor 1α Regulates a SOCS3-STAT3-Adiponectin Signal Transduction Pathway in Adipocytes
- (2012) Changtao Jiang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Desmodium gangeticum (Linn.) DC. Exhibits Antihypertrophic Effect in Isoproterenol-Induced Cardiomyoblasts via Amelioration of Oxidative Stress and Mitochondrial Alterations
- (2012) Vandana Sankar et al. JOURNAL OF CARDIOVASCULAR PHARMACOLOGY
- Curcumin Prevents High Fat Diet Induced Insulin Resistance and Obesity via Attenuating Lipogenesis in Liver and Inflammatory Pathway in Adipocytes
- (2012) Weijuan Shao et al. PLoS One
- Hypoxia-induced inflammatory cytokine secretion in human adipose tissue stromovascular cells
- (2011) R. W. O’Rourke et al. DIABETOLOGIA
- Regulation of hypoxia-inducible factor 1 and the loss of the cellular response to hypoxia in diabetes
- (2011) C. F. Bento et al. DIABETOLOGIA
- Adipose Tissue-specific Inhibition of Hypoxia-inducible Factor 1α Induces Obesity and Glucose Intolerance by Impeding Energy Expenditure in Mice
- (2010) Xinmei Zhang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- TNF-α induces mitochondrial dysfunction in 3T3-L1 adipocytes
- (2010) Xiao-Hui Chen et al. MOLECULAR AND CELLULAR ENDOCRINOLOGY
- Mitochondrial dysfunction is induced by high levels of glucose and free fatty acids in 3T3-L1 adipocytes
- (2010) Chun-Lin Gao et al. MOLECULAR AND CELLULAR ENDOCRINOLOGY
- Curcumin and obesity: evidence and mechanisms
- (2010) Lini Alappat et al. NUTRITION REVIEWS
- Adipose tissue failure and mitochondria as a possible target for improvement by bioactive food components
- (2009) Jaap Keijer et al. CURRENT OPINION IN LIPIDOLOGY
- Hypoxia-Inducible Factor 1 Induces Fibrosis and Insulin Resistance in White Adipose Tissue
- (2009) N. Halberg et al. MOLECULAR AND CELLULAR BIOLOGY
- Hypoxia stimulates lactate release and modulates monocarboxylate transporter (MCT1, MCT2, and MCT4) expression in human adipocytes
- (2009) Fátima Pérez de Heredia et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Cellular hypoxia and adipose tissue dysfunction in obesity
- (2009) I. Stuart Wood et al. PROCEEDINGS OF THE NUTRITION SOCIETY
- Role of hypoxia in obesity-induced disorders of glucose and lipid metabolism in adipose tissue
- (2008) Jun Yin et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Hypoxia Decreases Insulin Signaling Pathways in Adipocytes
- (2008) C. Regazzetti et al. DIABETES
- Curcumin and resveratrol inhibit nuclear factor-kappaB-mediated cytokine expression in adipocytes
- (2008) Amanda M Gonzales et al. Nutrition & Metabolism
- Hypoxia and the endocrine and signalling role of white adipose tissue
- (2008) Paul Trayhurn et al. ARCHIVES OF PHYSIOLOGY AND BIOCHEMISTRY
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now