An unexpected tail of VEGF and PlGF in pre-eclampsia

Impaired vascular permeability regulation caused by the VEGF165b splice variant in pre-eclampsia

(2011) VL Bills et al.   BJOG-AN INTERNATIONAL JOURNAL OF OBSTETRICS AND GYNAECOLOGY

Arginase contributes to endothelial cell oxidative stress in response to plasma from women with preeclampsia

(2009) Sowndramalingam Sankaralingam et al.   CARDIOVASCULAR RESEARCH

Dysregulation of anti-angiogenic agents (sFlt-1, PLGF, and sEndoglin) in preeclampsia—a step forward but not the definitive answer

(2009) J.M. Foidart et al.   JOURNAL OF REPRODUCTIVE IMMUNOLOGY

Alternatively spliced vascular endothelial growth factor receptor-2 is an essential endogenous inhibitor of lymphatic vessel growth

(2009) Romulo J C Albuquerque et al.   NATURE MEDICINE

Placental growth factor is a potent vasodilator of rat and human resistance arteries

(2008) George Osol et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Vascular Endothelial Growth Factor (VEGF)-A165b Is a Weak In vitro Agonist for VEGF Receptor-2 Due to Lack of Coreceptor Binding and Deficient Regulation of Kinase Activity

(2008) H. Kawamura et al.   CANCER RESEARCH

Failure to up-regulate VEGF165b in maternal plasma is a first trimester predictive marker for pre-eclampsia

(2008) Victoria L. Bills et al.   CLINICAL SCIENCE

Hypoxia is responsible for soluble vascular endothelial growth factor receptor-1 (VEGFR-1) but not for soluble endoglin induction in villous trophoblast

(2008) C. Munaut et al.   HUMAN REPRODUCTION