Myofibroblasts Exhibit Enhanced Fibronectin Assembly That Is Intrinsic to Their Contractile Phenotype

Inhibition of fibronectin deposition improves experimental liver fibrosis

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RPEL Proteins Are the Molecular Targets for CCG-1423, an Inhibitor of Rho Signaling

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Control of Myofibroblast Differentiation by Microtubule Dynamics through a Regulated Localization of mDia2

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Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis

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Matrix Stiffness–Induced Myofibroblast Differentiation Is Mediated by Intrinsic Mechanotransduction

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Quantitative microtiter fibronectin fibrillogenesis assay: use in high throughput screening for identification of inhibitor compounds

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NIH Image to ImageJ: 25 years of image analysis

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Delayed stress fiber formation mediates pulmonary myofibroblast differentiation in response to TGF-β

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Serum response factor is required for cell contact maintenance but dispensable for proliferation in visceral yolk sac endothelium

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Assembly of Fibronectin Extracellular Matrix

(2010) Purva Singh et al.   Annual Review of Cell and Developmental Biology

Extended Binding Site on Fibronectin for the Functional Upstream Domain of Protein F1 ofStreptococcus pyogenes

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Critical Role of Serum Response Factor in Pulmonary Myofibroblast Differentiation Induced by TGF-β

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Fibronectin Is an Important Regulator of Flow-Induced Vascular Remodeling

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Cell Traction Forces Direct Fibronectin Matrix Assembly

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Fibrillin Assembly Requires Fibronectin

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