Autophagy-dependent senescence in response to DNA damage and chronic apoptotic stress
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Autophagy-dependent senescence in response to DNA damage and chronic apoptotic stress
Authors
Keywords
-
Journal
Autophagy
Volume 8, Issue 2, Pages 236-251
Publisher
Informa UK Limited
Online
2012-03-05
DOI
10.4161/auto.8.2.18600
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis
- (2011) J. Y. Guo et al. GENES & DEVELOPMENT
- Interplay between oncogene-induced DNA damage response and heterochromatin in senescence and cancer
- (2011) Raffaella Di Micco et al. NATURE CELL BIOLOGY
- AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1
- (2011) Joungmok Kim et al. NATURE CELL BIOLOGY
- The role of the Atg1/ULK1 complex in autophagy regulation
- (2010) Noboru Mizushima CURRENT OPINION IN CELL BIOLOGY
- Regulation of the autophagy protein LC3 by phosphorylation
- (2010) Salvatore J. Cherra et al. JOURNAL OF CELL BIOLOGY
- A Cytoplasmic ATM-TRAF6-cIAP1 Module Links Nuclear DNA Damage Signaling to Ubiquitin-Mediated NF-κB Activation
- (2010) Michael Hinz et al. MOLECULAR CELL
- Autophagy and the Integrated Stress Response
- (2010) Guido Kroemer et al. MOLECULAR CELL
- Network organization of the human autophagy system
- (2010) Christian Behrends et al. NATURE
- Selective autophagy: ubiquitin-mediated recognition and beyond
- (2010) Claudine Kraft et al. NATURE CELL BIOLOGY
- Eaten alive: a history of macroautophagy
- (2010) Zhifen Yang et al. NATURE CELL BIOLOGY
- The Association of AMPK with ULK1 Regulates Autophagy
- (2010) Jong Woo Lee et al. PLoS One
- ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS
- (2010) A. Alexander et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Autophagy inhibition and antimalarials promote cell death in gastrointestinal stromal tumor (GIST)
- (2010) A. Gupta et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- ATM Activation by Oxidative Stress
- (2010) Z. Guo et al. SCIENCE
- Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy
- (2010) D. F. Egan et al. SCIENCE
- Poly(ADP-Ribose) Polymerase Inhibition as a Model for Synthetic Lethality in Developing Radiation Oncology Targets
- (2010) Anthony J. Chalmers et al. SEMINARS IN RADIATION ONCOLOGY
- p62 at the Crossroads of Autophagy, Apoptosis, and Cancer
- (2009) Jorge Moscat et al. CELL
- Hdm2 is a ubiquitin ligase of Ku70-Akt promotes cell survival by inhibiting Hdm2-dependent Ku70 destabilization
- (2009) V Gama et al. CELL DEATH AND DIFFERENTIATION
- Autophagy mediates the mitotic senescence transition
- (2009) A. R.J. Young et al. GENES & DEVELOPMENT
- ULK-Atg13-FIP200 Complexes Mediate mTOR Signaling to the Autophagy Machinery
- (2009) Chang Hwa Jung et al. MOLECULAR BIOLOGY OF THE CELL
- Promotion of autophagy as a mechanism for radiation sensitization of breast tumor cells
- (2009) David A. Gewirtz et al. RADIOTHERAPY AND ONCOLOGY
- The Atg8 and Atg12 ubiquitin-like conjugation systems in macroautophagy. ‘Protein Modifications: Beyond the Usual Suspects’ Review Series
- (2008) Jiefei Geng et al. EMBO REPORTS
- A C-terminal Fragment of Cyclin E, Generated by Caspase-mediated Cleavage, Is Degraded in the Absence of a Recognizable Phosphodegron
- (2008) Dragos Plesca et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Tumor Suppressor SMAR1 RepressesIκBα Expression and Inhibits p65 Transactivation through Matrix Attachment Regions
- (2008) Kamini Singh et al. JOURNAL OF BIOLOGICAL CHEMISTRY
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started