Folate Deficiency and FHIT Hypermethylation and HPV 16 Infection Promote Cervical Cancerization
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Title
Folate Deficiency and FHIT Hypermethylation and HPV 16 Infection Promote Cervical Cancerization
Authors
Keywords
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Journal
Asian Pacific Journal of Cancer Prevention
Volume 15, Issue 21, Pages 9313-9317
Publisher
Asian Pacific Organization for Cancer Prevention
Online
2015-02-24
DOI
10.7314/apjcp.2014.15.21.9313
References
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Note: Only part of the references are listed.- Assessment of the Prognostic Value of Methylation Status and Expression Levels of FHIT, GSTP1 and p16 in Non-Small Cell Lung Cancer in Egyptian Patients
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- A higher degree of LINE-1 methylation in peripheral blood mononuclear cells, a one-carbon nutrient related epigenetic alteration, is associated with a lower risk of developing cervical intraepithelial neoplasia
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- Promoter hypermethylation of RASSF1A, MGMT, and HIC-1 genes in benign and malignant colorectal tumors
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- Human papilloma virus persistence and neopterin, folate and homocysteine levels in cervical dysplasias
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- Common polymorphisms in methylenetetrahydrofolate reductase gene are associated with risks of cervical intraepithelial neoplasia and cervical cancer in women with low serum folate and vitamin B12
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- Folate Status and Aberrant DNA Methylation Are Associated With HPV Infection and Cervical Pathogenesis
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- Effect of aberrant promoter methylation ofFHITandRASSF1Agenes on susceptibility to cervical cancer in a North Indian population
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