Dual Oxidase 2 in Lung Epithelia Is Essential for Hyperoxia-Induced Acute Lung Injury in Mice
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Title
Dual Oxidase 2 in Lung Epithelia Is Essential for Hyperoxia-Induced Acute Lung Injury in Mice
Authors
Keywords
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Journal
ANTIOXIDANTS & REDOX SIGNALING
Volume 21, Issue 13, Pages 1803-1818
Publisher
Mary Ann Liebert Inc
Online
2014-04-27
DOI
10.1089/ars.2013.5677
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- A Key Role for NOX4 in Epithelial Cell Death During Development of Lung Fibrosis
- (2011) Stephanie Carnesecchi et al. ANTIOXIDANTS & REDOX SIGNALING
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- (2009) Horst Fischer ANTIOXIDANTS & REDOX SIGNALING
- Oxidative epithelial host defense is regulated by infectious and inflammatory stimuli
- (2009) Monica Valencia Gattas et al. FREE RADICAL BIOLOGY AND MEDICINE
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- (2009) David I. Brown et al. FREE RADICAL BIOLOGY AND MEDICINE
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- Multiple TLRs activate EGFR via a signaling cascade to produce innate immune responses in airway epithelium
- (2008) Jonathan L. Koff et al. AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY
- Role of Nox4 and Nox2 in Hyperoxia-Induced Reactive Oxygen Species Generation and Migration of Human Lung Endothelial Cells
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- NADPH oxidases in lung biology and pathology: Host defense enzymes, and more
- (2007) Albert van der Vliet FREE RADICAL BIOLOGY AND MEDICINE
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