p53-PGC-1α Pathway Mediates Oxidative Mitochondrial Damage and Cardiomyocyte Necrosis Induced by Monoamine Oxidase-A Upregulation: Role in Chronic Left Ventricular Dysfunction in Mice

Mitochondrial biogenesis in cardiac pathophysiology

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Telomere dysfunction induces metabolic and mitochondrial compromise

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p53 Regulates Oxidative Stress-Mediated Retrograde Signaling: A Novel Mechanism for Chemotherapy-Induced Cardiac Injury

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PGC-1α Regulates Expression of Myocardial Mitochondrial Antioxidants and Myocardial Oxidative Stress After Chronic Systolic Overload

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Glutathione Peroxidase-1 in Health and Disease: From Molecular Mechanisms to Therapeutic Opportunities

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Ca 2+ /Calmodulin-Dependent Kinase IIδ Causes Heart Failure by Accumulation of p53 in Dilated Cardiomyopathy

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Downregulation of catalase by reactive oxygen species via hypermethylation of CpG island II on the catalase promoter

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Disrupted Serotonergic and Sympathoadrenal Systems in Patients with Chronic Heart Failure May Serve as New Therapeutic Targets and Novel Biomarkers to Assess Severity, Progression and Response to Treatment

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Monoamine Oxidase A–Mediated Enhanced Catabolism of Norepinephrine Contributes to Adverse Remodeling and Pump Failure in Hearts With Pressure Overload

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Mutual Dependence of Foxo3a and PGC-1α in the Induction of Oxidative Stress Genes

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Genetic deletion of MAO-A promotes serotonin-dependent ventricular hypertrophy by pressure overload

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Use of pifithrin to inhibit p53-mediated signalling of TNF in dystrophic muscles of mdx mice

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The p53-cathepsin axis cooperates with ROS to activate programmed necrotic death upon DNA damage

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Mitochondrial oxidative stress and dysfunction in myocardial remodelling

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Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling

(2008) G. W. Dorn   CARDIOVASCULAR RESEARCH

Inhibition of GSK3β by Postconditioning Is Required to Prevent Opening of the Mitochondrial Permeability Transition Pore During Reperfusion

(2008) Ludovic Gomez et al.   CIRCULATION