Journal
ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
Volume 52, Issue 6, Pages 2009-2013Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/AAC.01594-07
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Funding
- NIAID NIH HHS [1 R43 AI069666-01, R43 AI069666] Funding Source: Medline
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Targeted antisense and gene replacement mutagenesis experiments demonstrate that only the murA1 gene and not the murA2 gene is required for the normal cellular growth of Bacillus anthracis. Antisense-based modulation of murA1 gene expression hypersensitizes cells to the MurA-specific antibiotic fosfomycin despite the normally high resistance of B. anthracis to this drug.
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