A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis

Nav1.8 channelopathy in mutant mice deficient for myelin protein zero is detrimental to motor axons

(2010) Mihai Moldovan et al.   BRAIN

Mechanisms Underlying Inflammation in Neurodegeneration

(2010) Christopher K. Glass et al.   CELL

Two Nedd4-binding Motifs Underlie Modulation of Sodium Channel Nav1.6 by p38 MAPK

(2010) Andreas Gasser et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Pharmacological management of symptoms in multiple sclerosis: current approaches and future directions

(2010) Alan J Thompson et al.   LANCET NEUROLOGY

CD45, CD148, and Lyp/Pep: critical phosphatases regulating Src family kinase signaling networks in immune cells

(2009) Michelle L. Hermiston et al.   IMMUNOLOGICAL REVIEWS

A robust and high-throughput Cre reporting and characterization system for the whole mouse brain

(2009) Linda Madisen et al.   NATURE NEUROSCIENCE

Distinct subsets of unmyelinated primary sensory fibers mediate behavioral responses to noxious thermal and mechanical stimuli

(2009) D. J. Cavanaugh et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Phosphorylation of Sodium Channel Nav1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons

(2008) A. Hudmon et al.   JOURNAL OF NEUROSCIENCE

Switch in the expression of mGlu1 and mGlu5 metabotropic glutamate receptors in the cerebellum of mice developing experimental autoimmune encephalomyelitis and in autoptic cerebellar samples from patients with multiple sclerosis

(2008) F. Fazio et al.   NEUROPHARMACOLOGY