Receptor for advanced glycation end-products (RAGE) is an indicator of direct lung injury in models of experimental lung injury

CD47 Deficiency Protects Mice from Lipopolysaccharide-Induced Acute Lung Injury and Escherichia coli Pneumonia

(2014) X. Su et al.   JOURNAL OF IMMUNOLOGY

Animal models of acute lung injury

(2008) Gustavo Matute-Bello et al.   AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY

Role of Soluble Receptor for Advanced Glycation End Products on Endotoxin-induced Lung Injury

(2008) Haiying Zhang et al.   AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE

A soluble form of the receptor for advanced glycation endproducts (RAGE) is produced by proteolytic cleavage of the membrane-bound form by the sheddase a disintegrin and metalloprotease 10 (ADAM10)

(2008) Angela Raucci et al.   FASEB JOURNAL

Expression of the receptor for advanced glycation end products in oligodendrocytes in response to oxidative stress

(2008) Jingdong Qin et al.   JOURNAL OF NEUROSCIENCE RESEARCH

Implication of receptor for advanced glycation end product (RAGE) in pulmonary health and pathophysiology

(2008) Tapan K. Mukherjee et al.   RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY

Plasma receptor for advanced glycation end products and clinical outcomes in acute lung injury

(2008) C S Calfee et al.   THORAX

Role of coagulation pathways and treatment with activated protein C in hyperoxic lung injury

(2008) M R Looney et al.   THORAX