Myofibroblasts cause heterogeneous Cx43 reduction and are unlikely to be coupled to myocytes in the healing canine infarct

Characterization of Gap Junction Remodeling in Epicardial Border Zone of Healing Canine Infarcts and Electrophysiological Effects of Partial Reversal by Rotigaptide

(2011) Ester Macia et al.   Circulation-Arrhythmia and Electrophysiology

Inhibition of renin-angiotensin system (RAS) reduces ventricular tachycardia risk by altering connexin43

(2011) Shahriar Iravanian et al.   JOURNAL OF MOLECULAR MEDICINE-JMM

Increased Cell–Cell Coupling Increases Infarct Size and Does not Decrease Incidence of Ventricular Tachycardia in Mice

(2011) Kevin A. Prestia et al.   Frontiers in Physiology

Structural and Molecular Mechanisms of Gap Junction Remodeling in Epicardial Border Zone Myocytes following Myocardial Infarction

(2009) Fabien Kieken et al.   CIRCULATION RESEARCH

Myofibroblasts in diseased hearts: New players in cardiac arrhythmias?

(2009) Stephan Rohr   HEART RHYTHM

Cell Therapy for Modification of the Myocardial Electrophysiological Substrate

(2008) Lior Yankelson et al.   CIRCULATION