Doxorubicin induces senescence or apoptosis in rat neonatal cardiomyocytes by regulating the expression levels of the telomere binding factors 1 and 2

Doxorubicin-induced mitochondrial dysfunction is secondary to nuclear p53 activation in H9c2 cardiomyoblasts

(2009) Vilma A. Sardão et al.   CANCER CHEMOTHERAPY AND PHARMACOLOGY

Trf1 Is Not Required for Proliferation or Functional Telomere Maintenance in Chicken DT40 Cells

(2009) Carol Cooley et al.   MOLECULAR BIOLOGY OF THE CELL

Induction of premature senescence in cardiomyocytes by doxorubicin as a novel mechanism of myocardial damage

(2008) Yasuhiro Maejima et al.   AGING CELL

Activation of Cardiac Progenitor Cells Reverses the Failing Heart Senescent Phenotype and Prolongs Lifespan

(2008) Arantxa Gonzalez et al.   CIRCULATION RESEARCH

Aging and Disease as Modifiers of Efficacy of Cell Therapy

(2008) Stefanie Dimmeler et al.   CIRCULATION RESEARCH

Doxorubicin, Cardiac Risk Factors, and Cardiac Toxicity in Elderly Patients With Diffuse B-Cell Non-Hodgkin's Lymphoma

(2008) Dawn L. Hershman et al.   JOURNAL OF CLINICAL ONCOLOGY

Microtubule Binding and Disruption and Induction of Premature Senescence by Disorazole C1

(2008) M. B. Tierno et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS

Notch1 regulates the fate of cardiac progenitor cells

(2008) A. Boni et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Telomere instability and cancer

(2007) J. Arturo Londoño-Vallejo   BIOCHIMIE