The functional −374T/A polymorphism of the receptor for advanced glycation end products may modulate Crohn's disease
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Title
The functional −374T/A polymorphism of the receptor for advanced glycation end products may modulate Crohn's disease
Authors
Keywords
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Journal
AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
Volume 300, Issue 5, Pages G823-G832
Publisher
American Physiological Society
Online
2011-02-11
DOI
10.1152/ajpgi.00115.2010
References
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- RAGE Gene Polymorphisms Are Associated with Circulating Levels of Endogenous Secretory RAGE but Not with Coronary Artery Disease in Chinese Patients with Type 2 Diabetes Mellitus
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- Serum levels of soluble receptor for advanced glycation end products and of S100 proteins are associated with inflammatory, autoantibody, and classical risk markers of joint and vascular damage in rheumatoid arthritis
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- A soluble form of the receptor for advanced glycation endproducts (RAGE) is produced by proteolytic cleavage of the membrane-bound form by the sheddase a disintegrin and metalloprotease 10 (ADAM10)
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- Receptor for Advanced Glycation End Products Is Subjected to Protein Ectodomain Shedding by Metalloproteinases
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- Glucocorticoids induce an activated, anti-inflammatory monocyte subset in mice that resembles myeloid-derived suppressor cells
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