Kcnj10 is a major type of K+ channel in mouse corneal epithelial cells and plays a role in initiating EGFR signaling

Src Family Protein Tyrosine Kinase Regulates the Basolateral K Channel in the Distal Convoluted Tubule (DCT) by Phosphorylation of KCNJ10 Protein

(2013) Chengbiao Zhang et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Ion channels and transporters in cancer. 1. Ion channels and cell proliferation in cancer

(2011) Andrea Becchetti   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Altered electroretinograms in patients with KCNJ10 mutations and EAST syndrome

(2011) Dorothy A. Thompson et al.   JOURNAL OF PHYSIOLOGY-LONDON

Inwardly Rectifying Potassium Channels: Their Structure, Function, and Physiological Roles

(2010) Hiroshi Hibino et al.   PHYSIOLOGICAL REVIEWS

Growth factors and corneal epithelial wound healing

(2009) Fu-Shin X. Yu et al.   BRAIN RESEARCH BULLETIN

Epilepsy, Ataxia, Sensorineural Deafness, Tubulopathy, andKCNJ10Mutations

(2009) Detlef Bockenhauer et al.   NEW ENGLAND JOURNAL OF MEDICINE

Seizures, sensorineural deafness, ataxia, mental retardation, and electrolyte imbalance (SeSAME syndrome) caused by mutations in KCNJ10

(2009) U. I. Scholl et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Functional implications for Kir4.1 channels in glial biology: from K+buffering to cell differentiation

(2008) Michelle L. Olsen et al.   JOURNAL OF NEUROCHEMISTRY