Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling

Direct observation of failing fibers in muscles of dystrophic mice provides mechanistic insight into muscular dystrophy

(2008) Dennis R. Claflin et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

The dynamics of the nitric oxide release-transient from stretched muscle cells

(2008) A.C. Wozniak et al.   INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY

TRPC1 binds to caveolin-3 and is regulated by Src kinase - role in Duchenne muscular dystrophy

(2008) O. L. Gervasio et al.   JOURNAL OF CELL SCIENCE

Between channels and tears: aim at ROS to save the membrane of dystrophic fibres

(2008) Carlo Reggiani   JOURNAL OF PHYSIOLOGY-LONDON

Comparison of the myoplasmic calcium transient elicited by an action potential in intact fibres ofmdxand normal mice

(2008) Stephen Hollingworth et al.   JOURNAL OF PHYSIOLOGY-LONDON

Mice Lacking Homer 1 Exhibit a Skeletal Myopathy Characterized by Abnormal Transient Receptor Potential Channel Activity

(2008) J. A. Stiber et al.   MOLECULAR AND CELLULAR BIOLOGY

Enhanced Excitation-Coupled Calcium Entry in Myotubes Expressing Malignant Hyperthermia Mutation R163C Is Attenuated by Dantrolene

(2008) G. Cherednichenko et al.   MOLECULAR PHARMACOLOGY

Muscle-specific overexpression of IGF-I improves E-C coupling in skeletal muscle fibers from dystrophic mdx mice

(2007) Jonathan D. Schertzer et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Reactive oxygen species contribute to Ca2+signals produced by osmotic stress in mouse skeletal muscle fibres

(2007) Adriano S. Martins et al.   JOURNAL OF PHYSIOLOGY-LONDON