Chronic Free-Choice Drinking in Crossed High Alcohol Preferring Mice Leads to Sustained Blood Ethanol Levels and Metabolic Tolerance Without Evidence of Liver Damage

Imipramine blocks ethanol-induced ASMase activation, ceramide generation, and PP2A activation, and ameliorates hepatic steatosis in ethanol-fed mice

(2011) Suthat Liangpunsakul et al.   AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY

Consilience of rodent and human phenotypes relevant for alcohol dependence

(2010)   ADDICTION BIOLOGY

Ethanol consumption: how should we measure it? Achieving consilience between human and animal phenotypes

(2010) Robert F. Leeman et al.   ADDICTION BIOLOGY

Laboratory Models Available to Study Alcohol-Induced Organ Damage and Immune Variations: Choosing the Appropriate Model

(2010) Nympha B. D’Souza El-Guindy et al.   ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH

Derivation and Characterization of Replicate High- and Low-Alcohol Preferring Lines of Mice and a High-Drinking Crossed HAP Line

(2010) Brandon Oberlin et al.   BEHAVIOR GENETICS

Ethanol drinking in rodents: is free-choice drinking related to the reinforcing effects of ethanol?

(2008) Alexis S. Green et al.   ALCOHOL

Supplementing the liquid alcohol diet with chow enhances alcohol intake in C57 BL/6 mice

(2008) A ANJI et al.   DRUG AND ALCOHOL DEPENDENCE