Chronic intermittent hypoxia-induced deficits in synaptic plasticity and neurocognitive functions: a role for brain-derived neurotrophic factor
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Title
Chronic intermittent hypoxia-induced deficits in synaptic plasticity and neurocognitive functions: a role for brain-derived neurotrophic factor
Authors
Keywords
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Journal
ACTA PHARMACOLOGICA SINICA
Volume 33, Issue 1, Pages 5-10
Publisher
Springer Nature
Online
2012-01-03
DOI
10.1038/aps.2011.184
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Note: Only part of the references are listed.- Intermittent Hypoxia-Induced Cognitive Deficits Are Mediated by NADPH Oxidase Activity in a Murine Model of Sleep Apnea
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- Intermittent Hypoxia Promotes Hippocampal Neurogenesis and Produces Antidepressant-Like Effects in Adult Rats
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- Brain-derived neurotrophic factor inhibits cell cycle reentry but not endoplasmic reticulum stress in cultured neurons following oxidative or excitotoxic stress
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- The Long-Term Health Plan and Disability Cost Benefit of Obstructive Sleep Apnea Treatment in a Commercial Motor Vehicle Driver Population
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- Brain-derived neurotrophic factor rescues and prevents chronic intermittent hypoxia-induced impairment of hippocampal long-term synaptic plasticity
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- Reactive oxygen species and the brain in sleep apnea
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- Daily intermittent hypoxia augments spinal BDNF levels, ERK phosphorylation and respiratory long-term facilitation
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- Sleep fragmentation reduces hippocampal CA1 pyramidal cell excitability and response to adenosine
- (2009) Jaime L. Tartar et al. NEUROSCIENCE LETTERS
- Up-regulating BDNF with an ampakine rescues synaptic plasticity and memory in Huntington's disease knockin mice
- (2009) D. A. Simmons et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Oxidative Stress—A Unifying Paradigm in Obstructive Sleep Apnea and Comorbidities
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- BDNF: A key regulator for protein synthesis-dependent LTP and long-term memory?
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- Effects of obstructive sleep apnea on cognitive function: A comparison between younger and older OSAS patients
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