MYC family amplification and clinical risk-factors interact to predict an extremely poor prognosis in childhood medulloblastoma

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Rapid Diagnosis of Medulloblastoma Molecular Subgroups

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Pleiotropic role for MYCN in medulloblastoma

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Adult and Pediatric Medulloblastomas Are Genetically Distinct and Require Different Algorithms for Molecular Risk Stratification

(2010) Andrey Korshunov et al.   JOURNAL OF CLINICAL ONCOLOGY

Integrative Genomic Analysis of Medulloblastoma Identifies a Molecular Subgroup That Drives Poor Clinical Outcome

(2010) Yoon-Jae Cho et al.   JOURNAL OF CLINICAL ONCOLOGY

Definition of Disease-Risk Stratification Groups in Childhood Medulloblastoma Using Combined Clinical, Pathologic, and Molecular Variables

(2010) David W. Ellison et al.   JOURNAL OF CLINICAL ONCOLOGY

Medulloblastoma Comprises Four Distinct Molecular Variants

(2010) Paul A. Northcott et al.   JOURNAL OF CLINICAL ONCOLOGY

Low-level copy number changes of MYC genes have a prognostic impact in medulloblastoma

(2010) Karel Zitterbart et al.   JOURNAL OF NEURO-ONCOLOGY

The potential impact of tumour biology on improved clinical practice for medulloblastoma: progress towards biologically driven clinical trials

(2009) Barry L. Pizer et al.   BRITISH JOURNAL OF NEUROSURGERY

The miR-17/92 Polycistron Is Up-regulated in Sonic Hedgehog-Driven Medulloblastomas and Induced by N-myc in Sonic Hedgehog-Treated Cerebellar Neural Precursors

(2009) P. A. Northcott et al.   CANCER RESEARCH

Outcome Prediction in Pediatric Medulloblastoma Based on DNA Copy-Number Aberrations of Chromosomes 6q and 17q and theMYCandMYCNLoci

(2009) Stefan Pfister et al.   JOURNAL OF CLINICAL ONCOLOGY

Gender affects survival for medulloblastoma only in older children and adults: A study from the surveillance epidemiology and end results registry

(2008) Emily K. Curran et al.   PEDIATRIC BLOOD & CANCER

Integrated Genomics Identifies Five Medulloblastoma Subtypes with Distinct Genetic Profiles, Pathway Signatures and Clinicopathological Features

(2008) Marcel Kool et al.   PLoS One