Elevated Expression of Serum Amyloid A 3 Protects Colon Epithelium Against Acute Injury Through TLR2-Dependent Induction of Neutrophil IL-22 Expression in a Mouse Model of Colitis
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Title
Elevated Expression of Serum Amyloid A 3 Protects Colon Epithelium Against Acute Injury Through TLR2-Dependent Induction of Neutrophil IL-22 Expression in a Mouse Model of Colitis
Authors
Keywords
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Journal
Frontiers in Immunology
Volume 9, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2018-06-29
DOI
10.3389/fimmu.2018.01503
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Note: Only part of the references are listed.- An IL-23R/IL-22 Circuit Regulates Epithelial Serum Amyloid A to Promote Local Effector Th17 Responses
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- Serum amyloid A1 isoforms display different efficacy at Toll-like receptor 2 and formyl peptide receptor 2
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- Intestinal Epithelial Serum Amyloid A Modulates Bacterial Growth In Vitro and Pro-Inflammatory Responses in Mouse Experimental Colitis
- (2010) Erik RM Eckhardt et al. BMC GASTROENTEROLOGY
- Invariant NKT cells modulate the suppressive activity of IL-10-secreting neutrophils differentiated with serum amyloid A
- (2010) Carmela De Santo et al. NATURE IMMUNOLOGY
- Serum amyloid A induces G-CSF expression and neutrophilia via Toll-like receptor 2
- (2008) R. L. He et al. BLOOD
- Innate and Adaptive Interleukin-22 Protects Mice from Inflammatory Bowel Disease
- (2008) Lauren A. Zenewicz et al. IMMUNITY
- IL-22 ameliorates intestinal inflammation in a mouse model of ulcerative colitis
- (2008) Ken Sugimoto et al. JOURNAL OF CLINICAL INVESTIGATION
- Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens
- (2008) Yan Zheng et al. NATURE MEDICINE
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