G9A promotes gastric cancer metastasis by upregulating ITGB3 in a SET domain-independent manner
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Title
G9A promotes gastric cancer metastasis by upregulating ITGB3 in a SET domain-independent manner
Authors
Keywords
-
Journal
Cell Death & Disease
Volume 9, Issue 3, Pages -
Publisher
Springer Nature
Online
2018-02-15
DOI
10.1038/s41419-018-0322-6
References
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Related references
Note: Only part of the references are listed.- Gastric cancer
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- G9a interacts with Snail and is critical for Snail-mediated E-cadherin repression in human breast cancer
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- Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response
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- G9a functions as a molecular scaffold for assembly of transcriptional coactivators on a subset of Glucocorticoid Receptor target genes
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- H3K9 Histone Methyltransferase G9a Promotes Lung Cancer Invasion and Metastasis by Silencing the Cell Adhesion Molecule Ep-CAM
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- Essential Role of the Histone Methyltransferase G9a in Cocaine-Induced Plasticity
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- Cancer stem cell-like SP cells have a high adhesion ability to the peritoneum in gastric carcinoma
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- Control of Cognition and Adaptive Behavior by the GLP/G9a Epigenetic Suppressor Complex
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- De novo DNA methylation promoted by G9a prevents reprogramming of embryonically silenced genes
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