The Role and Molecular Mechanism of Long Nocoding RNA-MEG3 in the Pathogenesis of Preeclampsia
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Title
The Role and Molecular Mechanism of Long Nocoding RNA-MEG3 in the Pathogenesis of Preeclampsia
Authors
Keywords
-
Journal
Reproductive Sciences
Volume -, Issue -, Pages 193371911774975
Publisher
SAGE Publications
Online
2018-01-24
DOI
10.1177/1933719117749753
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- (2014) Samy Lamouille et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
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- (2012) Aristidis Moustakas et al. SEMINARS IN CANCER BIOLOGY
- Accreta complicating complete placenta previa is characterized by reduced systemic levels of vascular endothelial growth factor and by epithelial-to-mesenchymal transition of the invasive trophoblast
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- (2011) Alicia M. Viloria-Petit et al. CELL CYCLE
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- (2011) York Hunt Ng et al. ENDOCRINOLOGY
- microRNA-29 can regulate expression of the long non-coding RNA gene MEG3 in hepatocellular cancer
- (2011) C Braconi et al. ONCOGENE
- Maternally Expressed Gene 3, an Imprinted Noncoding RNA Gene, Is Associated with Meningioma Pathogenesis and Progression
- (2010) X. Zhang et al. CANCER RESEARCH
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- (2010) Y. Zhou et al. DEVELOPMENT
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- (2010) M. Huarte et al. HUMAN MOLECULAR GENETICS
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- (2010) M.I. Kokkinos et al. PLACENTA
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- (2009) Tim R. Mercer et al. NATURE REVIEWS GENETICS
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- (2008) Berthold Huppertz HYPERTENSION
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