The effect of low and high-intensity cycling in diesel exhaust on flow-mediated dilation, circulating NOx, endothelin-1 and blood pressure
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Title
The effect of low and high-intensity cycling in diesel exhaust on flow-mediated dilation, circulating NOx, endothelin-1 and blood pressure
Authors
Keywords
Exercise, Nitric oxide, Blood plasma, Air pollution, Blood pressure, Arteries, Plasma volume, Nitrates
Journal
PLoS One
Volume 13, Issue 2, Pages e0192419
Publisher
Public Library of Science (PLoS)
Online
2018-02-23
DOI
10.1371/journal.pone.0192419
References
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Related references
Note: Only part of the references are listed.- Physiological Responses to Diesel Exhaust Exposure Are Modified by Cycling Intensity
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- Allometric scaling of diameter change in the original flow-mediated dilation protocol
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- Particulate Matter Air Pollution and Cardiovascular Disease
- (2010) Robert D. Brook et al. CIRCULATION
- eNOS uncoupling and endothelial dysfunction in aged vessels
- (2009) Yang-Ming Yang et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Impairment of coronary endothelial cell ETB receptor function after short-term inhalation exposure to whole diesel emissions
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- Contribution of Endothelin 1 to the Vascular Effects of Diesel Exhaust Inhalation in Humans
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- Pulmonary Nanoparticle Exposure Disrupts Systemic Microvascular Nitric Oxide Signaling
- (2009) Timothy R. Nurkiewicz et al. TOXICOLOGICAL SCIENCES
- The impact of baseline diameter on flow-mediated dilation differs in young and older humans
- (2008) Dick H. J. Thijssen et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Air Pollution Exposure Potentiates Hypertension Through Reactive Oxygen Species-Mediated Activation of Rho/ROCK
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- Diesel exhaust exposure enhances venoconstriction via uncoupling of eNOS
- (2008) Travis L. Knuckles et al. TOXICOLOGY AND APPLIED PHARMACOLOGY
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