Adipocyte-induced CD36 expression drives ovarian cancer progression and metastasis

Cancer treatment and survivorship statistics, 2016

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The Emerging Hallmarks of Cancer Metabolism

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Targeting metastasis-initiating cells through the fatty acid receptor CD36

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Integrative proteomic profiling of ovarian cancer cell lines reveals precursor cell associated proteins and functional status

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Genomics of Ovarian Cancer Progression Reveals Diverse Metastatic Trajectories Including Intraepithelial Metastasis to the Fallopian Tube

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Intratumor cholesteryl ester accumulation is associated with human breast cancer proliferation and aggressive potential: a molecular and clinicopathological study

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Obesity Contributes to Ovarian Cancer Metastatic Success through Increased Lipogenesis, Enhanced Vascularity, and Decreased Infiltration of M1 Macrophages

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Molecular Pathways: Trafficking of Metabolic Resources in the Tumor Microenvironment

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Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreatic adenocarcinoma

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Elevated free fatty acid uptake via CD36 promotes epithelial-mesenchymal transition in hepatocellular carcinoma

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Structure-Function of CD36 and Importance of Fatty Acid Signal Transduction in Fat Metabolism

(2014) Marta Yanina Pepino et al.   Annual Review of Nutrition

Regulation of AMPK Activation by CD36 Links Fatty Acid Uptake to β-Oxidation

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Mesothelial cells promote early ovarian cancer metastasis through fibronectin secretion

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Adipose tissue and adipocytes support tumorigenesis and metastasis

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Scavenger receptor CD36 mediates inhibition of cholesterol synthesis via activation of the PPARγ/PGC-1α pathway and Insig1/2 expression in hepatocytes

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Hypoxic and Ras-transformed cells support growth by scavenging unsaturated fatty acids from lysophospholipids

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Lipid metabolism in cancer

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Link between Intestinal CD36 Ligand Binding and Satiety Induced by a High Protein Diet in Mice

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Fatty acid transport proteins, implications in physiology and disease

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Adipocytes promote ovarian cancer metastasis and provide energy for rapid tumor growth

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Ovarian Cancer Development and Metastasis

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Crosstalk of reactive oxygen species and NF-κB signaling

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Targeting the Urokinase Plasminogen Activator Receptor Inhibits Ovarian Cancer Metastasis

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CD36 Is a Novel Serum Amyloid A (SAA) Receptor Mediating SAA Binding and SAA-induced Signaling in Human and Rodent Cells

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CD36 participates in a signaling pathway that regulates ROS formation in murine VSMCs

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Hypoxia Up-regulates CD36 Expression and Function via Hypoxia-inducible Factor-1- and Phosphatidylinositol 3-Kinase-dependent Mechanisms

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Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans

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Intra-abdominal tumor dissemination pattern and surgical outcome in 214 patients with primary ovarian cancer

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Fatty acid flux in adipocytes: The in's and out's of fat cell lipid trafficking

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CD36, a Scavenger Receptor Involved in Immunity, Metabolism, Angiogenesis, and Behavior

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Cellular fatty acid uptake: a pathway under construction

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Lipid Bodies Are Reservoirs of Cyclooxygenase-2 and Sites of Prostaglandin-E2 Synthesis in Colon Cancer Cells

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Novel Molecular Subtypes of Serous and Endometrioid Ovarian Cancer Linked to Clinical Outcome

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Loss of stearoyl-CoA desaturase 1 inhibits fatty acid oxidation and increases glucose utilization in the heart

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