NUDT21 negatively regulates PSMB2 and CXXC5 by alternative polyadenylation and contributes to hepatocellular carcinoma suppression
Published 2018 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
NUDT21 negatively regulates PSMB2 and CXXC5 by alternative polyadenylation and contributes to hepatocellular carcinoma suppression
Authors
Keywords
-
Journal
ONCOGENE
Volume -, Issue -, Pages -
Publisher
Springer Nature
Online
2018-05-17
DOI
10.1038/s41388-018-0280-6
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Alternative Polyadenylation in Triple-Negative Breast Tumors Allows NRAS and c-JUN to Bypass PUMILIO Posttranscriptional Regulation
- (2016) Wayne O. Miles et al. CANCER RESEARCH
- Alternative Polyadenylation: Another Foe in Cancer
- (2016) A. E. Erson-Bensan et al. MOLECULAR CANCER RESEARCH
- Alternative polyadenylation of mRNA precursors
- (2016) Bin Tian et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Hepatocellular carcinoma
- (2016) Josep M. Llovet et al. Nature Reviews Disease Primers
- Genome-wide profiling of polyadenylation sites reveals a link between selective polyadenylation and cancer metastasis
- (2015) D.-P. Lai et al. HUMAN MOLECULAR GENETICS
- NUDT21-spanning CNVs lead to neuropsychiatric disease and altered MeCP2 abundance via alternative polyadenylation
- (2015) Vincenzo A Gennarino et al. eLife
- CFIm25 links alternative polyadenylation to glioblastoma tumour suppression
- (2014) Chioniso P. Masamha et al. NATURE
- Time to evolve trial design after everolimus failure
- (2014) Josep M. Llovet Nature Reviews Clinical Oncology
- Alternative cleavage and polyadenylation: extent, regulation and function
- (2013) Ran Elkon et al. NATURE REVIEWS GENETICS
- Alternative Cleavage and Polyadenylation during Colorectal Cancer Development
- (2012) A. R. Morris et al. CLINICAL CANCER RESEARCH
- Hepatocellular carcinoma
- (2012) Alejandro Forner et al. LANCET
- Shortening of 3′UTRs Correlates with Poor Prognosis in Breast and Lung Cancer
- (2012) Antonio Lembo et al. PLoS One
- Genome-wide Analysis of Pre-mRNA 3′ End Processing Reveals a Decisive Role of Human Cleavage Factor I in the Regulation of 3′ UTR Length
- (2012) Georges Martin et al. Cell Reports
- RINF (CXXC5) is overexpressed in solid tumors and is an unfavorable prognostic factor in breast cancer†
- (2011) S. Knappskog et al. ANNALS OF ONCOLOGY
- Differential genome-wide profiling of tandem 3' UTRs among human breast cancer and normal cells by high-throughput sequencing
- (2011) Y. Fu et al. GENOME RESEARCH
- The Over-expression of the β2 Catalytic Subunit of the Proteasome Decreases Homologous Recombination and Impairs DNA Double-Strand Break Repair in Human Cells
- (2011) Anita Collavoli et al. JOURNAL OF BIOMEDICINE AND BIOTECHNOLOGY
- Mechanisms and Consequences of Alternative Polyadenylation
- (2011) Dafne Campigli Di Giammartino et al. MOLECULAR CELL
- Structural basis of UGUA recognition by the Nudix protein CFIm25 and implications for a regulatory role in mRNA 3' processing
- (2010) Q. Yang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Global Changes in Processing of mRNA 3' Untranslated Regions Characterize Clinically Distinct Cancer Subtypes
- (2009) P. Singh et al. CANCER RESEARCH
- Widespread Shortening of 3′UTRs by Alternative Cleavage and Polyadenylation Activates Oncogenes in Cancer Cells
- (2009) Christine Mayr et al. CELL
- Molecular Architecture of the Human Pre-mRNA 3′ Processing Complex
- (2009) Yongsheng Shi et al. MOLECULAR CELL
- Crystal structure of the 25 kDa subunit of human cleavage factor I m
- (2008) Molly Coseno et al. NUCLEIC ACIDS RESEARCH
- Proliferating Cells Express mRNAs with Shortened 3' Untranslated Regions and Fewer MicroRNA Target Sites
- (2008) R. Sandberg et al. SCIENCE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started