TMEM106B haplotypes have distinct gene expression patterns in aged brain
Published 2018 View Full Article
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Title
TMEM106B haplotypes have distinct gene expression patterns in aged brain
Authors
Keywords
TMEM106B, Frontotemporal dementia, Co-expression networks, Differential expression, Synaptic transmission, Immune response, Progranulin
Journal
Molecular Neurodegeneration
Volume 13, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-07-03
DOI
10.1186/s13024-018-0268-2
References
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- Reassessment of Risk Genotypes (GRN,TMEM106B, andABCC9Variants) Associated With Hippocampal Sclerosis of Aging Pathology
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- The TMEM106B locus and TDP-43 pathology in older persons without FTLD
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- VisANT 4.0: Integrative network platform to connect genes, drugs, diseases and therapies
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- Effect ofTMEM106BPolymorphism on Functional Network Connectivity in AsymptomaticGRNMutation Carriers
- (2013) Enrico Premi et al. JAMA Neurology
- TMEM106B risk variant is implicated in the pathologic presentation of Alzheimer disease
- (2012) N. J. Rutherford et al. NEUROLOGY
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- Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions
- (2010) Vivianna M Van Deerlin et al. NATURE GENETICS
- Common mechanisms in neurodegeneration
- (2010) Jesús Avila NATURE MEDICINE
- TMEM106B regulates progranulin levels and the penetrance of FTLD in GRN mutation carriers
- (2010) N. Finch et al. NEUROLOGY
- WGCNA: an R package for weighted correlation network analysis
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