An IFNγ/CXCL2 regulatory pathway determines lesion localization during EAE
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Title
An IFNγ/CXCL2 regulatory pathway determines lesion localization during EAE
Authors
Keywords
Experimental autoimmune encephalomyelitis, Multiple sclerosis, Chemokines, Cytokines, Neutrophils, Monocytes, Interferon-gamma
Journal
Journal of Neuroinflammation
Volume 15, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-07-16
DOI
10.1186/s12974-018-1237-y
References
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- Cytokine-Regulated Neutrophil Recruitment Is Required for Brain but Not Spinal Cord Inflammation during Experimental Autoimmune Encephalomyelitis
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- IFN-γ production by CD27+NK cells exacerbatesListeria monocytogenesinfection in mice by inhibiting granulocyte mobilization
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- Toll-like receptor (TLR) and inflammasome actions in the central nervous system
- (2012) Richa Hanamsagar et al. TRENDS IN IMMUNOLOGY
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- (2011) Veit Rothhammer et al. JOURNAL OF EXPERIMENTAL MEDICINE
- IL-23 modulated myelin-specific T cells induce EAE via an IFNγ driven, IL-17 independent pathway
- (2010) Mark A. Kroenke et al. BRAIN BEHAVIOR AND IMMUNITY
- EAE mediated by a non-IFN-γ/non-IL-17 pathway
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- Predominant Interferon-γ-Mediated Expression of CXCL9, CXCL10, and CCL5 Proteins in the Brain During Chronic Infection with Toxoplasma gondii in BALB/c Mice Resistant to Development of Toxoplasmic Encephalitis
- (2010) Xiangshu Wen et al. JOURNAL OF INTERFERON AND CYTOKINE RESEARCH
- C-C chemokine receptor 6–regulated entry of TH-17 cells into the CNS through the choroid plexus is required for the initiation of EAE
- (2009) Andrea Reboldi et al. NATURE IMMUNOLOGY
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- Differential regulation of central nervous system autoimmunity by TH1 and TH17 cells
- (2008) Ingunn M Stromnes et al. NATURE MEDICINE
- Macrophage activation by endogenous danger signals
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