Long noncoding RNA MALAT1 in exosomes drives regenerative function and modulates inflammation-linked networks following traumatic brain injury
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Title
Long noncoding RNA MALAT1 in exosomes drives regenerative function and modulates inflammation-linked networks following traumatic brain injury
Authors
Keywords
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Journal
Journal of Neuroinflammation
Volume 15, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-07-13
DOI
10.1186/s12974-018-1240-3
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Note: Only part of the references are listed.- Long Noncoding RNA Malat1 Regulates Cerebrovascular Pathologies in Ischemic Stroke
- (2017) Xuejing Zhang et al. JOURNAL OF NEUROSCIENCE
- The miR-200 family is increased in dysplastic lesions in ulcerative colitis patients
- (2017) Amy Lewis et al. PLoS One
- Induction of Multiple miR-200/182 Members in the Brains of Mice Are Associated with Acute Herpes Simplex Virus 1 Encephalitis
- (2017) Anna Majer et al. PLoS One
- Truncated EphA2 likely potentiates cell adhesion via integrins as well as infiltration and/or lodgment of a monocyte/macrophage cell line in the red pulp and marginal zone of the mouse spleen, where ephrin-A1 is prominently expressed in the vasculature
- (2016) Naoko Konda et al. HISTOCHEMISTRY AND CELL BIOLOGY
- MALAT1 and HOTAIR Long Non-Coding RNAs Play Opposite Role in Estrogen-Mediated Transcriptional Regulation in Prostate Cancer Cells
- (2016) Aurora Aiello et al. Scientific Reports
- 17β-Estradiol regulates cell proliferation, colony formation, migration, invasion and promotes apoptosis by upregulating miR-9 and thus degrades MALAT-1 in osteosarcoma cell MG-63 in an estrogen receptor-independent manner
- (2015) Dengfeng Fang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- miR-21 alleviates secondary blood–brain barrier damage after traumatic brain injury in rats
- (2015) Xintong Ge et al. BRAIN RESEARCH
- CCR2 Antagonism Alters Brain Macrophage Polarization and Ameliorates Cognitive Dysfunction Induced by Traumatic Brain Injury
- (2015) J. M. Morganti et al. JOURNAL OF NEUROSCIENCE
- Effect of exosomes derived from multipluripotent mesenchymal stromal cells on functional recovery and neurovascular plasticity in rats after traumatic brain injury
- (2015) Yanlu Zhang et al. JOURNAL OF NEUROSURGERY
- Chromatographically isolated CD63+CD81+extracellular vesicles from mesenchymal stromal cells rescue cognitive impairments after TBI
- (2015) Dong-ki Kim et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Hypoxia inducible factor-1 mediates expression of miR-322: potential role in proliferation and migration of pulmonary arterial smooth muscle cells
- (2015) Yan Zeng et al. Scientific Reports
- 17β-Estradiol treatment inhibits breast cell proliferation, migration and invasion by decreasing MALAT-1 RNA level
- (2014) Ziyi Zhao et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Small Nucleolar RNA Host Genes and Long Non-Coding RNA Responses in Directly Irradiated and Bystander Cells
- (2014) M. Ahmad Chaudhry CANCER BIOTHERAPY AND RADIOPHARMACEUTICALS
- lncRNA H19/miR-675 axis represses prostate cancer metastasis by targeting TGFBI
- (2014) Miaojun Zhu et al. FEBS Journal
- Exosomes as mediators of neuroinflammation
- (2014) Archana Gupta et al. Journal of Neuroinflammation
- Intravenous Transplants of Human Adipose-Derived Stem Cell Protect the Brain from Traumatic Brain Injury-Induced Neurodegeneration and Motor and Cognitive Impairments: Cell Graft Biodistribution and Soluble Factors in Young and Aged Rats
- (2014) N. Tajiri et al. JOURNAL OF NEUROSCIENCE
- Prognostic Impact of Bcl-2 Depends on Tumor Histology and Expression of MALAT-1 lncRNA in Non–Small-Cell Lung Cancer
- (2014) Lars Henning Schmidt et al. Journal of Thoracic Oncology
- The miR-545/374a Cluster Encoded in the Ftx lncRNA is Overexpressed in HBV-Related Hepatocellular Carcinoma and Promotes Tumorigenesis and Tumor Progression
- (2014) Qi Zhao et al. PLoS One
- Inflammation and white matter degeneration persist for years after a single traumatic brain injury
- (2013) Victoria E. Johnson et al. BRAIN
- miR-451 inhibits cell proliferation in human hepatocellular carcinoma through direct suppression of IKK-β
- (2013) He-Ping Li et al. CARCINOGENESIS
- miR-200 and miR-96 families repress neural induction from human embryonic stem cells
- (2013) Z.-W. Du et al. DEVELOPMENT
- Systemic Administration of Exosomes Released from Mesenchymal Stromal Cells Promote Functional Recovery and Neurovascular Plasticity After Stroke in Rats
- (2013) Hongqi Xin et al. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM
- Long-Term Upregulation of Inflammation and Suppression of Cell Proliferation in the Brain of Adult Rats Exposed to Traumatic Brain Injury Using the Controlled Cortical Impact Model
- (2013) Sandra A. Acosta et al. PLoS One
- The Antiapoptotic Function of miR-96 in Prostate Cancer by Inhibition of FOXO1
- (2013) Annika Fendler et al. PLoS One
- Exosomes: Current knowledge of their composition, biological functions, and diagnostic and therapeutic potentials
- (2012) Alexander V. Vlassov et al. BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS
- A Review of Neuroprotection Pharmacology and Therapies in Patients with Acute Traumatic Brain Injury
- (2012) Kevin W. McConeghy et al. CNS DRUGS
- Neuropathology of Explosive Blast Traumatic Brain Injury
- (2012) John Magnuson et al. Current Neurology and Neuroscience Reports
- Ly6C+Ly6G−Myeloid-derived suppressor cells play a critical role in the resolution of acute inflammation and the subsequent tissue repair process after spinal cord injury
- (2012) Hirokazu Saiwai et al. JOURNAL OF NEUROCHEMISTRY
- Intravenous multipotent adult progenitor cell therapy after traumatic brain injury: modulation of the resident microglia population
- (2012) Peter A Walker et al. Journal of Neuroinflammation
- Traumatic brain injury: networks and neuropathology
- (2012) Harvey Levin et al. LANCET NEUROLOGY
- The Neuropathology and Neurobiology of Traumatic Brain Injury
- (2012) Kaj Blennow et al. NEURON
- Bone marrow–derived stromal cell therapy for traumatic brain injury is neuroprotective via stimulation of non-neurologic organ systems
- (2012) Peter A. Walker et al. SURGERY
- Expression of miR-21, miR-31, miR-96 and miR-135b is correlated with the clinical parameters of colorectal cancer
- (2012) XIN MIN XU et al. Oncology Letters
- miR-96 regulates FOXO1-mediated cell apoptosis in bladder cancer
- (2012) YAN GUO et al. Oncology Letters
- miR-200bc/429 cluster modulates multidrug resistance of human cancer cell lines by targeting BCL2 and XIAP
- (2011) Wei Zhu et al. CANCER CHEMOTHERAPY AND PHARMACOLOGY
- Activation of ephrin A proteins influences hematopoietic stem cell adhesion and trafficking patterns
- (2010) Michael J. Ting et al. EXPERIMENTAL HEMATOLOGY
- The snoRNA MBII-52 (SNORD 115) is processed into smaller RNAs and regulates alternative splicing
- (2010) Shivendra Kishore et al. HUMAN MOLECULAR GENETICS
- Role of CCL2 (MCP-1) in Traumatic Brain Injury (TBI): Evidence from Severe TBI Patients and CCL2−/− Mice
- (2010) Bridgette D Semple et al. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM
- Deficiency of the chemokine receptor CXCR2 attenuates neutrophil infiltration and cortical damage following closed head injury
- (2010) Bridgette D. Semple et al. NEUROBIOLOGY OF DISEASE
- miR-10 in development and cancer
- (2009) A H Lund CELL DEATH AND DIFFERENTIATION
- Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites
- (2009) F. K. Swirski et al. SCIENCE
- snoTARGET shows that human orphan snoRNA targets locate close to alternative splice junctions
- (2007) Peter S. Bazeley et al. GENE
- 17β-Estradiol's salutary effects on splenic dendritic cell functions following trauma–hemorrhage are mediated via estrogen receptor-α
- (2007) Takashi Kawasaki et al. MOLECULAR IMMUNOLOGY
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