Role of dihydroartemisinin in regulating prostaglandin E2 synthesis cascade and inflammation in endothelial cells
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Title
Role of dihydroartemisinin in regulating prostaglandin E2 synthesis cascade and inflammation in endothelial cells
Authors
Keywords
Dihydroartemisinin, COX-1, COX-2, PGE<sub>2</sub>, Endothelial cells
Journal
HEART AND VESSELS
Volume -, Issue -, Pages -
Publisher
Springer Nature
Online
2018-05-23
DOI
10.1007/s00380-018-1190-9
References
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- (2010) Zhaojun Xiong et al. EUROPEAN JOURNAL OF PHARMACOLOGY
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