Lymphoblastoid cell lines as a model to understand amyotrophic lateral sclerosis disease mechanisms
Published 2018 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Lymphoblastoid cell lines as a model to understand amyotrophic lateral sclerosis disease mechanisms
Authors
Keywords
-
Journal
Disease Models & Mechanisms
Volume 11, Issue 3, Pages dmm031625
Publisher
The Company of Biologists
Online
2018-02-05
DOI
10.1242/dmm.031625
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- TDP-43 and FUS en route from the nucleus to the cytoplasm
- (2017) Helena Ederle et al. FEBS LETTERS
- TDP-43 and Cytoskeletal Proteins in ALS
- (2017) Moritz Oberstadt et al. MOLECULAR NEUROBIOLOGY
- Enhancing Mitofusin/Marf ameliorates neuromuscular dysfunction in Drosophila models of TDP-43 proteinopathies
- (2017) Bilal Khalil et al. NEUROBIOLOGY OF AGING
- Metabolic Dysfunctions in Amyotrophic Lateral Sclerosis Pathogenesis and Potential Metabolic Treatments
- (2017) Tesfaye W. Tefera et al. Frontiers in Neuroscience
- Inside out: the role of nucleocytoplasmic transport in ALS and FTLD
- (2016) Steven Boeynaems et al. ACTA NEUROPATHOLOGICA
- Mitochondria‐associated membrane collapse is a common pathomechanism in SIGMAR1 ‐ and SOD1 ‐linked ALS
- (2016) Seiji Watanabe et al. EMBO Molecular Medicine
- Decoding ALS: from genes to mechanism
- (2016) J. Paul Taylor et al. NATURE
- Early and gender-specific differences in spinal cord mitochondrial function and oxidative stress markers in a mouse model of ALS
- (2016) Daniel Cacabelos et al. Acta Neuropathologica Communications
- Gene-specific mitochondria dysfunctions in human TARDBP and C9ORF72 fibroblasts
- (2016) Elisa Onesto et al. Acta Neuropathologica Communications
- In vitro studies in VCP-associated multisystem proteinopathy suggest altered mitochondrial bioenergetics
- (2015) Angèle Nalbandian et al. MITOCHONDRION
- Altered age-related changes in bioenergetic properties and mitochondrial morphology in fibroblasts from sporadic amyotrophic lateral sclerosis patients
- (2015) Scott P. Allen et al. NEUROBIOLOGY OF AGING
- Overexpression of nuclear FUS induces neuronal cell death
- (2015) H. Suzuki et al. NEUROSCIENCE
- Nonnative SOD1 trimer is toxic to motor neurons in a model of amyotrophic lateral sclerosis
- (2015) Elizabeth A. Proctor et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- FUS Interacts with HSP60 to Promote Mitochondrial Damage
- (2015) Jianwen Deng et al. PLoS Genetics
- Mitochondria dynamism: of shape, transport and cell migration
- (2014) André Ferreira da Silva et al. CELLULAR AND MOLECULAR LIFE SCIENCES
- Disturbed mitochondrial dynamics and neurodegenerative disorders
- (2014) Florence Burté et al. Nature Reviews Neurology
- The ALS disease-associated mutant TDP-43 impairs mitochondrial dynamics and function in motor neurons
- (2013) Wenzhang Wang et al. HUMAN MOLECULAR GENETICS
- Abnormal mitochondrial transport and morphology are common pathological denominators in SOD1 and TDP43 ALS mouse models
- (2013) Jordi Magrané et al. HUMAN MOLECULAR GENETICS
- The changing scene of amyotrophic lateral sclerosis
- (2013) Wim Robberecht et al. NATURE REVIEWS NEUROSCIENCE
- Altered Intracellular Localization of SOD1 in Leukocytes from Patients with Sporadic Amyotrophic Lateral Sclerosis
- (2013) Cristina Cereda et al. PLoS One
- Morphological abnormalities in mitochondria of the skin of patients with sporadic amyotrophic lateral sclerosis
- (2012) Gabriel E. Rodríguez et al. ARQUIVOS DE NEURO-PSIQUIATRIA
- Mutant SOD1G93A triggers mitochondrial fragmentation in spinal cord motor neurons: Neuroprotection by SIRT3 and PGC-1α
- (2012) Wenjun Song et al. NEUROBIOLOGY OF DISEASE
- An over-oxidized form of superoxide dismutase found in sporadic amyotrophic lateral sclerosis with bulbar onset shares a toxic mechanism with mutant SOD1
- (2012) S. Guareschi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Mitochondrial dynamics in neurodegeneration
- (2012) Kie Itoh et al. TRENDS IN CELL BIOLOGY
- Protein Misdirection Inside and Outside Motor Neurons in Amyotrophic Lateral Sclerosis (ALS): A Possible Clue for Therapeutic Strategies
- (2011) Akemi Ido et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Amyotrophic Lateral Sclerosis Multiprotein Biomarkers in Peripheral Blood Mononuclear Cells
- (2011) Giovanni Nardo et al. PLoS One
- TDP-43 and FUS: a nuclear affair
- (2011) Dorothee Dormann et al. TRENDS IN NEUROSCIENCES
- Wild-Type Human TDP-43 Expression Causes TDP-43 Phosphorylation, Mitochondrial Aggregation, Motor Deficits, and Early Mortality in Transgenic Mice
- (2010) Y.-F. Xu et al. JOURNAL OF NEUROSCIENCE
- Mitochondrial dysfunction and intracellular calcium dysregulation in ALS
- (2010) Hibiki Kawamata et al. MECHANISMS OF AGEING AND DEVELOPMENT
- SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
- (2010) Stella Gagliardi et al. NEUROBIOLOGY OF DISEASE
- In Vivo Accumulation of Helicobacter pylori Products, NOD1, Ubiquitinated Proteins and Proteasome in a Novel Cytoplasmic Structure
- (2010) Vittorio Necchi et al. PLoS One
- Non–cell autonomous toxicity in neurodegenerative disorders: ALS and beyond
- (2009) Hristelina Ilieva et al. JOURNAL OF CELL BIOLOGY
- Focal degeneration of astrocytes in amyotrophic lateral sclerosis
- (2008) D Rossi et al. CELL DEATH AND DIFFERENTIATION
- Skeletal Muscle Is a Primary Target of SOD1G93A-Mediated Toxicity
- (2008) Gabriella Dobrowolny et al. Cell Metabolism
- Fission and selective fusion govern mitochondrial segregation and elimination by autophagy
- (2008) Gilad Twig et al. EMBO JOURNAL
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAdd your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload Now