p73 coordinates with Δ133p53 to promote DNA double-strand break repair
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Title
p73 coordinates with Δ133p53 to promote DNA double-strand break repair
Authors
Keywords
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Journal
CELL DEATH AND DIFFERENTIATION
Volume 25, Issue 6, Pages 1063-1079
Publisher
Springer Nature
Online
2018-03-06
DOI
10.1038/s41418-018-0085-8
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Note: Only part of the references are listed.- Increased Δ133p53 mRNA in lung carcinoma corresponds with reduction of p21 expression
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- p53 isoform Δ113p53/Δ133p53 promotes DNA double-strand break repair to protect cell from death and senescence in response to DNA damage
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- TAp73β-mediated suppression of cell migration requires p57Kip2 control of actin cytoskeleton dynamics
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- p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes
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- Development of Novel Visual-Plus Quantitative Analysis Systems for Studying DNA Double-Strand Break Repairs in Zebrafish
- (2012) Jingang Liu et al. Journal of Genetics and Genomics
- The p53 isoform, Δ133p53α, stimulates angiogenesis and tumour progression
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- Δ133p53 is an independent prognostic marker in p53 mutant advanced serous ovarian cancer
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- Genome-Wide Mapping Indicates That p73 and p63 Co-Occupy Target Sites and Have Similar DNA-Binding Profiles In Vivo
- (2010) Annie Yang et al. PLoS One
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- Overexpression of RAD51 suppresses recombination defects: a possible mechanism to reverse genomic instability
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- (2009) Yu-Li Lin et al. PLoS Genetics
- TAp73 knockout shows genomic instability with infertility and tumor suppressor functions
- (2008) R. Tomasini et al. GENES & DEVELOPMENT
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