Telmisartan protects chronic intermittent hypoxic mice via modulating cardiac renin-angiotensin system activity

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Short-term use of telmisartan attenuates oxidation and improves Prdx2 expression more than antioxidant β-blockers in the cardiovascular systems of spontaneously hypertensive rats

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Early apoptosis in different models of cardiac hypertrophy induced by high renin-angiotensin system activity involves CaMKII

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Effect of Telmisartan on local cardiovascular oxidative stress in mouse under chronic intermittent hypoxia condition

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Local angiotensin II aggravates cardiac remodeling in hypertension

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Angiotensin II Up-Regulates Angiotensin I-Converting Enzyme (ACE), but Down-Regulates ACE2 via the AT1-ERK/p38 MAP Kinase Pathway

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Sleep Apnea and Cardiovascular Disease

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Increased Expression of Angiotensin Converting Enzyme 2 in Conjunction with Reduction of Neointima by Angiotensin II Type 1 Receptor Blockade

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