Relaxin protects cardiac muscle cells from hypoxia/reoxygenation injury: involvement of the Notch-1 pathway
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Title
Relaxin protects cardiac muscle cells from hypoxia/reoxygenation injury: involvement of the Notch-1 pathway
Authors
Keywords
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Journal
FASEB JOURNAL
Volume 29, Issue 1, Pages 239-249
Publisher
FASEB
Online
2014-10-24
DOI
10.1096/fj.14-254854
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- Notch signaling activation contributes to cardioprotection provided by ischemic preconditioning and postconditioning
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- Serelaxin, recombinant human relaxin-2, for treatment of acute heart failure (RELAX-AHF): a randomised, placebo-controlled trial
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- Relaxin promotes growth and maturation of mouse neonatal cardiomyocytes in vitro: clues for cardiac regeneration
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- Relaxin remodels fibrotic healing following myocardial infarction
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- Novel therapeutic concepts * The epidemic of cardiovascular disease in the developing world: global implications
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- Skeletal myoblasts overexpressing relaxin improve differentiation and communication of primary murine cardiomyocyte cell cultures
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- Activation of Notch-Mediated Protective Signaling in the Myocardium
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- Functional and histopathological improvement of the post-infarcted rat heart upon myoblast cell grafting and relaxin therapy
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- The multifaceted role of Notch in cardiac development and disease
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